Marcos F. Fondevila

Santiago De Compostela, GA 15782, Spain

Last Updated: 09/26/2026

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Found 61 publications

Non-absorbable antibiotics worsen alcohol-associated liver disease in gastric acid-suppressed mice.

Non-absorbable antibiotics worsen alcohol-associated liver disease in gastric acid-suppressed mice.

Journal: Gut microbes

Published: July 09, 2026

Targeting microbial bile salt hydrolase reprograms bile acid metabolism and ameliorates metabolic dysfunction-associated steatohepatitis in mice.

Targeting microbial bile salt hydrolase reprograms bile acid metabolism and ameliorates metabolic dysfunction-associated steatohepatitis in mice.

Journal: Molecular metabolism

Published: June 01, 2026

Targeting Microbial Bile Salt Hydrolase Reprograms Bile Acid Metabolism and Ameliorates Metabolic Dysfunction-Associated Steatohepatitis in Mice.

Targeting Microbial Bile Salt Hydrolase Reprograms Bile Acid Metabolism and Ameliorates Metabolic Dysfunction-Associated Steatohepatitis in Mice.

Journal: bioRxiv : the preprint server for biology

Published: May 25, 2026

Reply to: "Hepatic CTSB is Kupffer cell-enriched, whereas disease-state stratification is more strongly captured by endotoxin/TLR4 programmes".

Reply to: "Hepatic CTSB is Kupffer cell-enriched, whereas disease-state stratification is more strongly captured by endotoxin/TLR4 programmes".

Journal: Journal of hepatology

Published: April 23, 2026

Faecal Cathepsin B and S Are Associated With Liver Disease Severity and Adiposity in MASLD.

Faecal Cathepsin B and S Are Associated With Liver Disease Severity and Adiposity in MASLD.

Journal: Liver international : official journal of the International Association for the Study of the Liver

Published: March 01, 2026

Parallel regulation of goblet cell-associated antigen passages by reduced microbial sensing and mAChR4 signaling in Muc2 deficiency prevents ethanol-induced liver injury.

Parallel regulation of goblet cell-associated antigen passages by reduced microbial sensing and mAChR4 signaling in Muc2 deficiency prevents ethanol-induced liver injury.

Journal: Hepatology (Baltimore, Md.)

Published: September 17, 2025

Macrophage-derived cathepsin B disrupts intestinal tight junctions through occludin degradation and promotes alcohol-associated liver disease.

Macrophage-derived cathepsin B disrupts intestinal tight junctions through occludin degradation and promotes alcohol-associated liver disease.

Journal: Journal of hepatology

Published: July 18, 2025

Macrophage-derived cathepsin B disrupts intestinal tight junctions through occludin degradation and promotes alcohol-associated liver disease.

Macrophage-derived cathepsin B disrupts intestinal tight junctions through occludin degradation and promotes alcohol-associated liver disease.

Journal: Journal of hepatology

Published: July 18, 2025

Peptide YY reduces cytotoxicity of Candida albicans in alcohol-associated liver disease.

Peptide YY reduces cytotoxicity of Candida albicans in alcohol-associated liver disease.

Journal: JHEP reports : innovation in hepatology

Published: April 15, 2025

Activation of intestinal endogenous retroviruses by alcohol exacerbates liver disease.

Activation of intestinal endogenous retroviruses by alcohol exacerbates liver disease.

Journal: The Journal of clinical investigation

Published: October 31, 2024

Targeted inhibition of pathobiont virulence factor mitigates alcohol-associated liver disease.

Targeted inhibition of pathobiont virulence factor mitigates alcohol-associated liver disease.

Journal: Cell host & microbe

Published: September 29, 2024

Lack of PCK1 in hepatic stellate cells causes liver fibrosis by fueling tricarboxylic acid cycle and increasing glycolysis.

Lack of PCK1 in hepatic stellate cells causes liver fibrosis by fueling tricarboxylic acid cycle and increasing glycolysis.

Journal: Cell metabolism

Published: August 06, 2024

Showing 1-12 of 61

Last Updated: 09/26/2026