Marcos Matamoros
Madrid, MD, Spain
Last Updated: 09/26/2026
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Found 11 publications
The p.P888L SAP97 polymorphism increases the transient outward current (Ito,f) and abbreviates the action potential duration and the QT interval.
Journal: Scientific reports
Published: December 26, 2019
Cardiac Kir2.1 and NaV1.5 Channels Traffic Together to the Sarcolemma to Control Excitability.
Journal: Circulation research
Published: August 09, 2017
Kir2.1-Nav1.5 Channel Complexes Are Differently Regulated than Kir2.1 and Nav1.5 Channels Alone.
Journal: Frontiers in physiology
Published: July 26, 2017
Brugada syndrome trafficking-defective Nav1.5 channels can trap cardiac Kir2.1/2.2 channels.
Journal: JCI insight
Published: July 14, 2017
Tbx20 controls the expression of the KCNH2 gene and of hERG channels.
Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: January 05, 2017
Nav1.5 N-terminal domain binding to α1-syntrophin increases membrane density of human Kir2.1, Kir2.2 and Nav1.5 channels.
Journal: Cardiovascular research
Published: September 21, 2015
Structural basis of drugs that increase cardiac inward rectifier Kir2.1 currents.
Journal: Cardiovascular research
Published: September 11, 2014
Chronic atrial fibrillation increases microRNA-21 in human atrial myocytes decreasing L-type calcium current.
Journal: Circulation. Arrhythmia and electrophysiology
Published: August 10, 2014
Propafenone blocks human cardiac Kir2.x channels by decreasing the negative electrostatic charge in the cytoplasmic pore.
Journal: Biochemical pharmacology
Published: January 18, 2013
Showing 1-11 of 11
Last Updated: 09/26/2026