Maria J. Manzano-Andres
Madrid, MD, Spain
Last Updated: 09/26/2026
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Found 20 publications
Lamin A/C Expression in Hematopoietic Cells Declines During Human Aging and Constrains Atherosclerosis in Mice.
Journal: Arteriosclerosis, thrombosis, and vascular biology
Published: July 03, 2025
Exacerbated atherosclerosis in progeria is prevented by progerin elimination in vascular smooth muscle cells but not endothelial cells.
Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: April 22, 2024
Endothelial YAP/TAZ activation promotes atherosclerosis in a mouse model of Hutchinson-Gilford progeria syndrome.
Journal: The Journal of clinical investigation
Published: July 06, 2023
Lamin A/C Ablation Restricted to Vascular Smooth Muscle Cells, Cardiomyocytes, and Cardiac Fibroblasts Causes Cardiac and Vascular Dysfunction.
Journal: International journal of molecular sciences
Published: June 16, 2023
Atherosclerotic plaque development in mice is enhanced by myeloid ZEB1 downregulation.
Journal: Nature communications
Published: January 25, 2023
Premature Vascular Aging with Features of Plaque Vulnerability in an Atheroprone Mouse Model of Hutchinson-Gilford Progeria Syndrome with Ldlr Deficiency.
Journal: Cells
Published: August 12, 2020
Paclitaxel mitigates structural alterations and cardiac conduction system defects in a mouse model of Hutchinson-Gilford progeria syndrome.
Journal: Cardiovascular research
Published: June 01, 2020
Last Updated: 09/26/2026