Maria L. Pedrosa-Vera
Madrid, MD, Spain
Last Updated: 09/26/2026
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Found 12 publications
Cardiac Macrophages and Fibroblasts Modulate Atrial Fibrillation Maintenance.
Journal: Circulation research
Published: February 10, 2026
Cardiac Macrophages and Fibroblasts Modulate Atrial Fibrillation Maintenance.
Journal: Circulation research
Published: February 10, 2026
Kir2.1 mutations differentially increase the risk of flecainide proarrhythmia in Andersen Tawil Syndrome.
Journal: medRxiv : the preprint server for health sciences
Published: December 23, 2024
Extracellular Kir2.1C122Y Mutant Upsets Kir2.1-PIP2 Bonds and Is Arrhythmogenic in Andersen-Tawil Syndrome.
Journal: Circulation research
Published: March 18, 2024
The Kir2.1E299V mutation increases atrial fibrillation vulnerability while protecting the ventricles against arrhythmias in a mouse model of short QT syndrome type 3.
Journal: Cardiovascular research
Published: July 04, 2023
Extracellular cysteine disulfide bond break at Cys122 disrupts PIP2-dependent Kir2.1 channel function and leads to arrhythmias in Andersen-Tawil Syndrome.
Journal: bioRxiv : the preprint server for biology
Published: June 19, 2023
Intermediate molecular phenotypes to identify genetic markers of anthracycline-induced cardiotoxicity risk.
Journal: bioRxiv : the preprint server for biology
Published: January 30, 2023
Kir2.1 dysfunction at the sarcolemma and the sarcoplasmic reticulum causes arrhythmias in a mouse model of Andersen-Tawil syndrome type 1.
Journal: Nature cardiovascular research
Published: March 28, 2022
Molecular stratification of arrhythmogenic mechanisms in the Andersen Tawil syndrome.
Journal: Cardiovascular research
Published: March 18, 2022
SNTA1 gene rescues ion channel function and is antiarrhythmic in cardiomyocytes derived from induced pluripotent stem cells from muscular dystrophy patients.
Journal: eLife
Published: December 21, 2021
Showing 1-12 of 12
Last Updated: 09/26/2026