Maria Souza-Oliveira
1524 Prof. Lineu Prestes Avenue
Sao Paulo, SP 05508000, Brazil
Sao Paulo, SP 05508000, Brazil
Last Updated: 09/26/2026
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Found 16 publications
Exogenous uromodulin and tubular responses in a model of oxalate-induced kidney injury.
Journal: American journal of physiology. Renal physiology
Published: September 02, 2025
Renal ischemia-reperfusion injury triggers proximal tubular apoptosis and NHE3 dysfunction via p38MAPK/ezrin signaling pathway.
Journal: American journal of physiology. Renal physiology
Published: July 03, 2025
Insights into the effects of apelin-13 on renal function and NHE3 activity following ischemia/reperfusion-induced acute kidney injury.
Journal: Frontiers in physiology
Published: December 12, 2024
Renal ischemia/reperfusion induces prominent progressive kidney disease in diabetic mice.
Journal: American journal of physiology. Endocrinology and metabolism
Published: July 17, 2024
Early type 1 diabetes aggravates renal ischemia/reperfusion-induced acute kidney injury.
Journal: Scientific reports
Published: January 12, 2021
Long-Term Angiotensin II Infusion Induces Oxidative and Endoplasmic Reticulum Stress and Modulates Na+ Transporters Through the Nephron.
Journal: Frontiers in physiology
Published: December 16, 2020
Sodium Oxalate-Induced Acute Kidney Injury Associated With Glomerular and Tubulointerstitial Damage in Rats.
Journal: Frontiers in physiology
Published: April 06, 2020
Intracellular albumin overload elicits endoplasmic reticulum stress and PKC-delta/p38 MAPK pathway activation to induce podocyte apoptosis.
Journal: Scientific reports
Published: April 16, 2018
Angiotensin II-induced podocyte apoptosis is mediated by endoplasmic reticulum stress/PKC-δ/p38 MAPK pathway activation and trough increased Na+/H+ exchanger isoform 1 activity.
Journal: BMC nephrology
Published: August 28, 2017
The Role of β-Adrenergic Overstimulation in the Early Stages of Renal Injury.
Journal: Kidney & blood pressure research
Published: June 28, 2017
Beta-2-microglobulin (B2M) expression in the urinary sediment correlates with clinical markers of kidney disease in patients with type 1 diabetes.
Journal: Metabolism: clinical and experimental
Published: November 10, 2015
Last Updated: 09/26/2026