Mariangela Storto

Milan, Italy

Last Updated: 09/26/2026

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Found 10 publications

Iron overload damages mitochondria and induces metabolic rewiring of hematopoietic stem cells toward glycolysis.

Iron overload damages mitochondria and induces metabolic rewiring of hematopoietic stem cells toward glycolysis.

Journal: Blood
Published: September 09, 2025

Imbalanced TGFβ signalling and autophagy drive erythroid priming of hematopoietic stem cells in β-thalassemia.

Imbalanced TGFβ signalling and autophagy drive erythroid priming of hematopoietic stem cells in β-thalassemia.

Journal: Nature communications
Published: June 02, 2024

Inhibition of FGF23 is a therapeutic strategy to target hematopoietic stem cell niche defects in β-thalassemia.

Inhibition of FGF23 is a therapeutic strategy to target hematopoietic stem cell niche defects in β-thalassemia.

Journal: Science translational medicine
Published: May 31, 2023

Dietary intake of cyanidin-3-glucoside induces a long-lasting cardioprotection from ischemia/reperfusion injury by altering the microbiota.

Dietary intake of cyanidin-3-glucoside induces a long-lasting cardioprotection from ischemia/reperfusion injury by altering the microbiota.

Journal: The Journal of nutritional biochemistry
Published: December 16, 2020

Heme catabolism by tumor-associated macrophages controls metastasis formation.

Heme catabolism by tumor-associated macrophages controls metastasis formation.

Journal: Nature immunology
Published: January 27, 2020

Tumor-Derived Prostaglandin E2 Promotes p50 NF-κB-Dependent Differentiation of Monocytic MDSCs.

Tumor-Derived Prostaglandin E2 Promotes p50 NF-κB-Dependent Differentiation of Monocytic MDSCs.

Journal: Cancer research
Published: September 11, 2019

Hematopoietic stem cell function in β-thalassemia is impaired and is rescued by targeting the bone marrow niche.

Hematopoietic stem cell function in β-thalassemia is impaired and is rescued by targeting the bone marrow niche.

Journal: Blood
Published: July 31, 2019

Nicotinamide Phosphoribosyltransferase Acts as a Metabolic Gate for Mobilization of Myeloid-Derived Suppressor Cells.

Nicotinamide Phosphoribosyltransferase Acts as a Metabolic Gate for Mobilization of Myeloid-Derived Suppressor Cells.

Journal: Cancer research
Published: May 19, 2018

Metabolic influence on the differentiation of suppressive myeloid cells in cancer.

Metabolic influence on the differentiation of suppressive myeloid cells in cancer.

Journal: Carcinogenesis
Published: April 11, 2018

Cinnamic anilides as new mitochondrial permeability transition pore inhibitors endowed with ischemia-reperfusion injury protective effect in vivo.

Cinnamic anilides as new mitochondrial permeability transition pore inhibitors endowed with ischemia-reperfusion injury protective effect in vivo.

Journal: Journal of medicinal chemistry
Published: June 12, 2014
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Last Updated: 09/26/2026