Marie-elodie Cattin
Croissy-sur-seine, 78290, France
Last Updated: 09/26/2026
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Found 8 publications
Expression of murine muscle-enriched A-type lamin-interacting protein (MLIP) is regulated by tissue-specific alternative transcription start sites.
Journal: The Journal of biological chemistry
Published: May 14, 2018
Filling the drug discovery gap: is high-content screening the missing link?
Journal: Current opinion in pharmacology
Published: May 03, 2018
In Vitro Modeling of Congenital Heart Defects Associated with an NKX2-5 Mutation Revealed a Dysregulation in BMP/Notch-Mediated Signaling.
Journal: Stem cells (Dayton, Ohio)
Published: February 24, 2017
Mutation in lamin A/C sensitizes the myocardium to exercise-induced mechanical stress but has no effect on skeletal muscles in mouse.
Journal: Neuromuscular disorders : NMD
Published: April 17, 2016
Deletion of MLIP (muscle-enriched A-type lamin-interacting protein) leads to cardiac hyperactivation of Akt/mammalian target of rapamycin (mTOR) and impaired cardiac adaptation.
Journal: The Journal of biological chemistry
Published: July 13, 2015
Heterozygous LmnadelK32 mice develop dilated cardiomyopathy through a combined pathomechanism of haploinsufficiency and peptide toxicity.
Journal: Human molecular genetics
Published: April 12, 2013
'State-of-the-heart' of cardiac laminopathies.
Journal: Current opinion in cardiology
Published: March 05, 2013
DelK32-lamin A/C has abnormal location and induces incomplete tissue maturation and severe metabolic defects leading to premature death.
Journal: Human molecular genetics
Published: November 18, 2011
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Last Updated: 09/26/2026