Mark P. Bond

Bristol, ENG BS28HW, United Kingdom

Last Updated: 09/26/2026

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Found 28 publications

In Silico Discovery and Characterization of a Novel Nuclear Transcription Factor-Y (NF-Y) Inhibitor with Antimitogenic Properties.

In Silico Discovery and Characterization of a Novel Nuclear Transcription Factor-Y (NF-Y) Inhibitor with Antimitogenic Properties.

Journal: Journal of medicinal chemistry

Published: March 20, 2026

Extracellular matrix stiffness primes the NLRP3-Inflammasome by promoting NLRP3 gene expression via FAK/mTOR/nuclear-actin/SREBP signalling in vascular smooth muscle cells.

Extracellular matrix stiffness primes the NLRP3-Inflammasome by promoting NLRP3 gene expression via FAK/mTOR/nuclear-actin/SREBP signalling in vascular smooth muscle cells.

Journal: Biochimica et biophysica acta. Molecular cell research

Published: March 16, 2026

Epigenetic regulation of TIMP-3 expression in human macrophages via physiological and pharmacological modification of histone3 lysine27 (H3K27).

Epigenetic regulation of TIMP-3 expression in human macrophages via physiological and pharmacological modification of histone3 lysine27 (H3K27).

Journal: Molecular biology reports

Published: May 16, 2025

Extracellular matrix stiffness controls cardiac fibroblast proliferation via the nuclear factor-Y (NF-Y) transcription factor.

Extracellular matrix stiffness controls cardiac fibroblast proliferation via the nuclear factor-Y (NF-Y) transcription factor.

Journal: Biochimica et biophysica acta. Molecular cell research

Published: August 31, 2023

Substrate stiffness promotes vascular smooth muscle cell calcification by reducing the levels of nuclear actin monomers.

Substrate stiffness promotes vascular smooth muscle cell calcification by reducing the levels of nuclear actin monomers.

Journal: Journal of molecular and cellular cardiology

Published: July 27, 2023

Combined role for YAP-TEAD and YAP-RUNX2 signalling in substrate-stiffness regulation of cardiac fibroblast proliferation.

Combined role for YAP-TEAD and YAP-RUNX2 signalling in substrate-stiffness regulation of cardiac fibroblast proliferation.

Journal: Biochimica et biophysica acta. Molecular cell research

Published: March 11, 2022

Cyclic-AMP Increases Nuclear Actin Monomer Which Promotes Proteasomal Degradation of RelA/p65 Leading to Anti-Inflammatory Effects.

Cyclic-AMP Increases Nuclear Actin Monomer Which Promotes Proteasomal Degradation of RelA/p65 Leading to Anti-Inflammatory Effects.

Journal: Cells

Published: January 18, 2022

Epigenetic Regulation of F2RL3 Associates With Myocardial Infarction and Platelet Function.

Epigenetic Regulation of F2RL3 Associates With Myocardial Infarction and Platelet Function.

Journal: Circulation research

Published: January 11, 2022

Nuclear actin regulates cell proliferation and migration via inhibition of SRF and TEAD.

Nuclear actin regulates cell proliferation and migration via inhibition of SRF and TEAD.

Journal: Biochimica et biophysica acta. Molecular cell research

Published: November 14, 2019

Ending Restenosis: Inhibition of Vascular Smooth Muscle Cell Proliferation by cAMP.

Ending Restenosis: Inhibition of Vascular Smooth Muscle Cell Proliferation by cAMP.

Journal: Cells

Published: October 24, 2019

Elevated cyclic-AMP represses expression of exchange protein activated by cAMP (EPAC1) by inhibiting YAP-TEAD activity and HDAC-mediated histone deacetylation.

Elevated cyclic-AMP represses expression of exchange protein activated by cAMP (EPAC1) by inhibiting YAP-TEAD activity and HDAC-mediated histone deacetylation.

Journal: Biochimica et biophysica acta. Molecular cell research

Published: March 06, 2019

Antiproliferative and Antimigratory Effects of a Novel YAP-TEAD Interaction Inhibitor Identified Using in Silico Molecular Docking.

Antiproliferative and Antimigratory Effects of a Novel YAP-TEAD Interaction Inhibitor Identified Using in Silico Molecular Docking.

Journal: Journal of medicinal chemistry

Published: January 15, 2019

Showing 1-12 of 28

Last Updated: 09/26/2026