Marlene Dufresne
Toulouse, France
Last Updated: 09/26/2026
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Found 16 publications
The E3 ubiquitin ligase TRIP12 is required for pancreatic acinar cell plasticity and pancreatic carcinogenesis.
Journal: The Journal of pathology
Published: November 14, 2023
Upregulated Apelin Signaling in Pancreatic Cancer Activates Oncogenic Signaling Pathways to Promote Tumor Development.
Journal: International journal of molecular sciences
Published: July 28, 2022
E3 Ubiquitin Ligase TRIP12: Regulation, Structure, and Physiopathological Functions.
Journal: International journal of molecular sciences
Published: September 25, 2020
The E3 ubiquitin ligase TRIP12 participates in cell cycle progression and chromosome stability.
Journal: Scientific reports
Published: September 23, 2019
Experimental pancreatic cancer develops in soft pancreas: novel leads for an individualized diagnosis by ultrafast elasticity imaging.
Journal: Theranostics
Published: February 13, 2019
First-in-man phase 1 clinical trial of gene therapy for advanced pancreatic cancer: safety, biodistribution, and preliminary clinical findings.
Journal: Molecular therapy : the journal of the American Society of Gene Therapy
Published: December 09, 2014
Pancreatic cell plasticity and cancer initiation induced by oncogenic Kras is completely dependent on wild-type PI 3-kinase p110α.
Journal: Genes & development
Published: December 03, 2014
The E3 ubiquitin ligase thyroid hormone receptor-interacting protein 12 targets pancreas transcription factor 1a for proteasomal degradation.
Journal: The Journal of biological chemistry
Published: October 31, 2014
Loss of Somatostatin Receptor Subtype 2 Promotes Growth of KRAS-Induced Pancreatic Tumors in Mice by Activating PI3K Signaling and Overexpression of CXCL16.
Journal: Gastroenterology
Published: April 11, 2014
Pancreatic preneoplastic lesions plasma signatures and biomarkers based on proteome profiling of mouse models.
Journal: British journal of cancer
Published: March 27, 2014
Last Updated: 09/26/2026