Melissa A. Pruski

Internal Medicine
1 Genesys Pkwy
Grand Blanc, MI 48439

Last Updated: 09/26/2026

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Found 8 publications

CD73-Dependent Adenosine Signaling through Adora2b Drives Immunosuppression in Ductal Pancreatic Cancer.

CD73-Dependent Adenosine Signaling through Adora2b Drives Immunosuppression in Ductal Pancreatic Cancer.

Journal: Cancer research
Published: August 15, 2022

Correction to: Kras mutation rate precisely orchestrates ductal derived pancreatic intraepithelial neoplasia and pancreatic cancer.

Correction to: Kras mutation rate precisely orchestrates ductal derived pancreatic intraepithelial neoplasia and pancreatic cancer.

Journal: Laboratory investigation; a journal of technical methods and pathology
Published: June 03, 2021

Kras mutation rate precisely orchestrates ductal derived pancreatic intraepithelial neoplasia and pancreatic cancer.

Kras mutation rate precisely orchestrates ductal derived pancreatic intraepithelial neoplasia and pancreatic cancer.

Journal: Laboratory investigation; a journal of technical methods and pathology
Published: June 03, 2020

Mst1/2 kinases restrain transformation in a novel transgenic model of Ras driven non-small cell lung cancer.

Mst1/2 kinases restrain transformation in a novel transgenic model of Ras driven non-small cell lung cancer.

Journal: Oncogene
Published: December 17, 2018

Mutant p53R175H promotes cancer initiation in the pancreas by stabilizing HSP70.

Mutant p53R175H promotes cancer initiation in the pancreas by stabilizing HSP70.

Journal: Cancer letters
Published: November 12, 2018

Immune Cell Production of Interleukin 17 Induces Stem Cell Features of Pancreatic Intraepithelial Neoplasia Cells.

Immune Cell Production of Interleukin 17 Induces Stem Cell Features of Pancreatic Intraepithelial Neoplasia Cells.

Journal: Gastroenterology
Published: May 30, 2017

p53 mutations cooperate with oncogenic Kras to promote adenocarcinoma from pancreatic ductal cells.

p53 mutations cooperate with oncogenic Kras to promote adenocarcinoma from pancreatic ductal cells.

Journal: Oncogene
Published: June 20, 2015

Posttraumatic stress disorder-like induction elevates β-amyloid levels, which directly activates corticotropin-releasing factor neurons to exacerbate stress responses.

Posttraumatic stress disorder-like induction elevates β-amyloid levels, which directly activates corticotropin-releasing factor neurons to exacerbate stress responses.

Journal: The Journal of neuroscience : the official journal of the Society for Neuroscience
Published: February 13, 2015
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Last Updated: 09/26/2026