Melissa N. Van Tok
Amsterdam, NH 1105AZ, Netherlands
Last Updated: 09/26/2026
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Found 15 publications
Dual Blockade of TNF and IL-17A Inhibits Inflammation and Structural Damage in a Rat Model of Spondyloarthritis.
Journal: International journal of molecular sciences
Published: December 13, 2021
Paradoxical Augmentation of Experimental Spondyloarthritis by RORC Inhibition in HLA-B27 Transgenic Rats.
Journal: Frontiers in immunology
Published: April 24, 2021
Transmembrane TNF drives osteoproliferative joint inflammation reminiscent of human spondyloarthritis.
Journal: The Journal of experimental medicine
Published: February 17, 2020
Interleukin-17A Inhibition Diminishes Inflammation and New Bone Formation in Experimental Spondyloarthritis.
Journal: Arthritis & rheumatology (Hoboken, N.J.)
Published: January 29, 2018
Innate Immune Activation Can Trigger Experimental Spondyloarthritis in HLA-B27/Huβ2m Transgenic Rats.
Journal: Frontiers in immunology
Published: May 30, 2017
The Transcriptional Coactivator Bob1 Is Associated With Pathologic B Cell Responses in Autoimmune Tissue Inflammation.
Journal: Arthritis & rheumatology (Hoboken, N.J.)
Published: July 10, 2016
Insulin-Like Growth Factor I Does Not Drive New Bone Formation in Experimental Arthritis.
Journal: PloS one
Published: April 26, 2016
Data showing non-conventional HLA-B27 expression in axial joints and gut tissue from B27 transgenic rats, and in frozen and paraffin-fixed synovial SpA tissue.
Journal: Data in brief
Published: March 15, 2016
Non-conventional forms of HLA-B27 are expressed in spondyloarthritis joints and gut tissue.
Journal: Journal of autoimmunity
Published: February 11, 2016
Interleukin-9 Overexpression and Th9 Polarization Characterize the Inflamed Gut, the Synovial Tissue, and the Peripheral Blood of Patients With Psoriatic Arthritis.
Journal: Arthritis & rheumatology (Hoboken, N.J.)
Published: July 01, 2015
Last Updated: 09/26/2026