Meng Hao

Jiangsu Key Laboratory Of New Drug Research And Clinical Pharmacy, 209 Tongshan Road
Xuzhou, 221004, China

Last Updated: 09/26/2026

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Found 10 publications

Correction to: Novel biphenyl diester derivative AB-38b inhibits NLRP3 inflammasome through Nrf2 activation in diabetic nephropathy.

Correction to: Novel biphenyl diester derivative AB-38b inhibits NLRP3 inflammasome through Nrf2 activation in diabetic nephropathy.

Journal: Cell biology and toxicology

Published: February 16, 2022

Novel Chalcone BDD-39 Mitigated Diabetic Nephropathy through the Activation of Nrf2/ARE Signaling.

Novel Chalcone BDD-39 Mitigated Diabetic Nephropathy through the Activation of Nrf2/ARE Signaling.

Journal: Current molecular pharmacology

Published: November 04, 2020

YAP mediates the interaction between the Hippo and PI3K/Akt pathways in mesangial cell proliferation in diabetic nephropathy.

YAP mediates the interaction between the Hippo and PI3K/Akt pathways in mesangial cell proliferation in diabetic nephropathy.

Journal: Acta diabetologica

Published: March 07, 2020

Correction to: novel biphenyl diester derivative AB-38b inhibits NLRP3 inflammasome through Nrf2 activation in diabetic nephropathy.

Correction to: novel biphenyl diester derivative AB-38b inhibits NLRP3 inflammasome through Nrf2 activation in diabetic nephropathy.

Journal: Cell biology and toxicology

Published: January 24, 2020

Keap1/Nrf2/ARE signaling unfolds therapeutic targets for redox imbalanced-mediated diseases and diabetic nephropathy.

Keap1/Nrf2/ARE signaling unfolds therapeutic targets for redox imbalanced-mediated diseases and diabetic nephropathy.

Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

Published: August 16, 2019

Novel biphenyl diester derivative AB-38b inhibits NLRP3 inflammasome through Nrf2 activation in diabetic nephropathy.

Novel biphenyl diester derivative AB-38b inhibits NLRP3 inflammasome through Nrf2 activation in diabetic nephropathy.

Journal: Cell biology and toxicology

Published: July 23, 2019

A novel compound AB38b attenuates oxidative stress and ECM protein accumulation in kidneys of diabetic mice through modulation of Keap1/Nrf2 signaling.

A novel compound AB38b attenuates oxidative stress and ECM protein accumulation in kidneys of diabetic mice through modulation of Keap1/Nrf2 signaling.

Journal: Acta pharmacologica Sinica

Published: April 08, 2019

The mTOR promotes oxidative stress-induced apoptosis of mesangial cells in diabetic nephropathy.

The mTOR promotes oxidative stress-induced apoptosis of mesangial cells in diabetic nephropathy.

Journal: Molecular and cellular endocrinology

Published: June 15, 2017

Quercetin inhibited epithelial mesenchymal transition in diabetic rats, high-glucose-cultured lens, and SRA01/04 cells through transforming growth factor-β2/phosphoinositide 3-kinase/Akt pathway.

Quercetin inhibited epithelial mesenchymal transition in diabetic rats, high-glucose-cultured lens, and SRA01/04 cells through transforming growth factor-β2/phosphoinositide 3-kinase/Akt pathway.

Journal: Molecular and cellular endocrinology

Published: December 20, 2016

Antidiabetic cataract effects of GbE, rutin and quercetin are mediated by the inhibition of oxidative stress and polyol pathway.

Antidiabetic cataract effects of GbE, rutin and quercetin are mediated by the inhibition of oxidative stress and polyol pathway.

Journal: Acta biochimica Polonica

Published: July 07, 2016

Showing 1-10 of 10

Last Updated: 09/26/2026