Mengying Wu

Union Hospital
Wuhan, 430022, China

Last Updated: 09/26/2026

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Found 13 publications

Phase separation-based HTS identifies cobimetinib as a YAP-TEAD inhibitor that suppresses hyperactivated YAP-induced cancer progression.

Phase separation-based HTS identifies cobimetinib as a YAP-TEAD inhibitor that suppresses hyperactivated YAP-induced cancer progression.

Journal: Science translational medicine

Published: April 22, 2026

Correction: Plasma Exosomal miR-199a-5p Derived from Preeclampsia with Severe Features Impairs Endothelial Cell Function via Targeting SIRT1.

Correction: Plasma Exosomal miR-199a-5p Derived from Preeclampsia with Severe Features Impairs Endothelial Cell Function via Targeting SIRT1.

Journal: Reproductive sciences (Thousand Oaks, Calif.)

Published: December 12, 2023

The tumor suppressor NF2 modulates TEAD4 stability and activity in Hippo signaling via direct interaction.

The tumor suppressor NF2 modulates TEAD4 stability and activity in Hippo signaling via direct interaction.

Journal: The Journal of biological chemistry

Published: December 10, 2023

Corrigendum to PVT1/miR-145-5p/HK2 modulates vascular smooth muscle cells phenotype switch via glycolysis: The new perspective on the spiral artery remodeling [Placenta 130 (2022) 25-33].

Corrigendum to PVT1/miR-145-5p/HK2 modulates vascular smooth muscle cells phenotype switch via glycolysis: The new perspective on the spiral artery remodeling [Placenta 130 (2022) 25-33].

Journal: Placenta

Published: December 10, 2022

Placenta-Targeted Nanoparticles Loaded with PFKFB3 Overexpression Plasmids Enhance Angiogenesis and Placental Function.

Placenta-Targeted Nanoparticles Loaded with PFKFB3 Overexpression Plasmids Enhance Angiogenesis and Placental Function.

Journal: Bioengineering (Basel, Switzerland)

Published: September 21, 2022

PVT1/miR-145-5p/HK2 modulates vascular smooth muscle cells phenotype switch via glycolysis: The new perspective on the spiral artery remodeling.

PVT1/miR-145-5p/HK2 modulates vascular smooth muscle cells phenotype switch via glycolysis: The new perspective on the spiral artery remodeling.

Journal: Placenta

Published: May 02, 2022

Plasma Exosomal miR-199a-5p Derived from Preeclampsia with Severe Features Impairs Endothelial Cell Function via Targeting SIRT1.

Plasma Exosomal miR-199a-5p Derived from Preeclampsia with Severe Features Impairs Endothelial Cell Function via Targeting SIRT1.

Journal: Reproductive sciences (Thousand Oaks, Calif.)

Published: January 27, 2022

Pathogenic mechanisms of preeclampsia with severe features implied by the plasma exosomal mirna profile.

Pathogenic mechanisms of preeclampsia with severe features implied by the plasma exosomal mirna profile.

Journal: Bioengineered

Published: October 26, 2021

Metformin Corrects Glucose Metabolism Reprogramming and NLRP3 Inflammasome-Induced Pyroptosis via Inhibiting the TLR4/NF-κB/PFKFB3 Signaling in Trophoblasts: Implication for a Potential Therapy of Preeclampsia.

Metformin Corrects Glucose Metabolism Reprogramming and NLRP3 Inflammasome-Induced Pyroptosis via Inhibiting the TLR4/NF-κB/PFKFB3 Signaling in Trophoblasts: Implication for a Potential Therapy of Preeclampsia.

Journal: Oxidative medicine and cellular longevity

Published: June 28, 2021

PFKFB3 regulates lipopolysaccharide-induced excessive inflammation and cellular dysfunction in HTR-8/Svneo cells: Implications for the role of PFKFB3 in preeclampsia.

PFKFB3 regulates lipopolysaccharide-induced excessive inflammation and cellular dysfunction in HTR-8/Svneo cells: Implications for the role of PFKFB3 in preeclampsia.

Journal: Placenta

Published: November 19, 2020

MALAT1 sponges miR-26a and miR-26b to regulate endothelial cell angiogenesis via PFKFB3-driven glycolysis in early-onset preeclampsia.

MALAT1 sponges miR-26a and miR-26b to regulate endothelial cell angiogenesis via PFKFB3-driven glycolysis in early-onset preeclampsia.

Journal: Molecular therapy. Nucleic acids

Published: June 16, 2020

Structural analysis of fungal CENP-H/I/K homologs reveals a conserved assembly mechanism underlying proper chromosome alignment.

Structural analysis of fungal CENP-H/I/K homologs reveals a conserved assembly mechanism underlying proper chromosome alignment.

Journal: Nucleic acids research

Published: June 25, 2018

Showing 1-12 of 13

Last Updated: 09/26/2026