Meredith Hanna

Hamilton, ON, Canada

Last Updated: 09/26/2026

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Found 12 publications

Hb F-SickKids (HBG2: C.308A > G): A Novel γ-Globin Variant Associated with Transient Neonatal Cyanosis.

Hb F-SickKids (HBG2: C.308A > G): A Novel γ-Globin Variant Associated with Transient Neonatal Cyanosis.

Journal: Hemoglobin
Published: June 10, 2026

Novel δβ Fusion Gene in Individuals of Bhutanese and Nepalese Origin.

Novel δβ Fusion Gene in Individuals of Bhutanese and Nepalese Origin.

Journal: International journal of laboratory hematology
Published: March 04, 2026

Newborn Screening for β-Thalassemia Identifies a Complex Genotype Involving a Novel β-Globin Gene Mutation (HBB:c.336dup).

Newborn Screening for β-Thalassemia Identifies a Complex Genotype Involving a Novel β-Globin Gene Mutation (HBB:c.336dup).

Journal: Hemoglobin
Published: April 02, 2024

β0-Thalassemia Caused by a Novel Nonsense Mutation [HBB:c.199A > T].

β0-Thalassemia Caused by a Novel Nonsense Mutation [HBB:c.199A > T].

Journal: Hemoglobin
Published: March 01, 2024

Splice Acceptor Mutation [HBB:c.93-2A > T] in a Patient with Hb S/β0-Thalassemia.

Splice Acceptor Mutation [HBB:c.93-2A > T] in a Patient with Hb S/β0-Thalassemia.

Journal: Hemoglobin
Published: February 15, 2024

Characterization of Two Novel Deletions Involving the 5' Region of the β-Globin Gene.

Characterization of Two Novel Deletions Involving the 5' Region of the β-Globin Gene.

Journal: Hemoglobin
Published: November 29, 2017

α0-Thalassemia Due to a 90.7 kb Deletion (- -NFLD).

α0-Thalassemia Due to a 90.7 kb Deletion (- -NFLD).

Journal: Hemoglobin
Published: August 26, 2017

Novel Mutation of the Translation Initiation Codon of the α1-Globin Gene (ATG>AAG or HBA1:c.2T>A).

Novel Mutation of the Translation Initiation Codon of the α1-Globin Gene (ATG>AAG or HBA1:c.2T>A).

Journal: Hemoglobin
Published: November 09, 2016

Sudanese (δβ)0-Thalassemia: Identification and Characterization of a Novel 9.6 kb Deletion.

Sudanese (δβ)0-Thalassemia: Identification and Characterization of a Novel 9.6 kb Deletion.

Journal: Hemoglobin
Published: July 09, 2015

α(+)-Thalassemia Due to a Frameshift Mutation of the α2-Globin Gene [codons 55/56 (+T) or HBA2: c.168dup].

α(+)-Thalassemia Due to a Frameshift Mutation of the α2-Globin Gene [codons 55/56 (+T) or HBA2: c.168dup].

Journal: Hemoglobin
Published: April 22, 2015

Hb S/β+-thalassemia due to Hb sickle and a novel deletion of DNase I hypersensitive sites HS3 and HS4 of the β locus control region.

Hb S/β+-thalassemia due to Hb sickle and a novel deletion of DNase I hypersensitive sites HS3 and HS4 of the β locus control region.

Journal: Haematologica
Published: February 16, 2015

Non-thalassemic phenotype associated with the -83 (G > A) mutation of the β-globin gene promoter (HBB: c.-133G > A).

Non-thalassemic phenotype associated with the -83 (G > A) mutation of the β-globin gene promoter (HBB: c.-133G > A).

Journal: Hemoglobin
Published: November 19, 2014
Showing 1-12 of 12

Last Updated: 09/26/2026