Merve Bolat

Erzurum, 25200, Turkey

Last Updated: 09/26/2026

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Found 28 publications

Rosmarinic Acid Attenuates Isoniazid-Induced Neurotoxicity by Restoring Mitochondrial Dynamics and Suppressing Oxidative Stress, Ferroptosis, Neuroinflammation, and Apoptosis.

Rosmarinic Acid Attenuates Isoniazid-Induced Neurotoxicity by Restoring Mitochondrial Dynamics and Suppressing Oxidative Stress, Ferroptosis, Neuroinflammation, and Apoptosis.

Journal: Tissue & cell

Published: July 10, 2026

Morin attenuates vancomycin-induced nephrotoxicity via Nrf-2/HO-1, SIRT1/PGC1α and AKT/FOXO1A pathways.

Morin attenuates vancomycin-induced nephrotoxicity via Nrf-2/HO-1, SIRT1/PGC1α and AKT/FOXO1A pathways.

Journal: Molecular biology reports

Published: July 02, 2026

p-Coumaric Acid Attenuates Lead Acetate-induced Neurotoxicity in Rats by Improving Behavioral Dysfunction and Suppressing Oxidative Stress, Neuroinflammation, Apoptosis, and Plasticity-related Molecular Alterations.

p-Coumaric Acid Attenuates Lead Acetate-induced Neurotoxicity in Rats by Improving Behavioral Dysfunction and Suppressing Oxidative Stress, Neuroinflammation, Apoptosis, and Plasticity-related Molecular Alterations.

Journal: Biological trace element research

Published: May 08, 2026

Selenium attenuates paclitaxel-induced peripheral neuropathy by suppressing oxidative stress, inflammation, and apoptosis in rat sciatic nerve.

Selenium attenuates paclitaxel-induced peripheral neuropathy by suppressing oxidative stress, inflammation, and apoptosis in rat sciatic nerve.

Journal: Neurotoxicology

Published: February 25, 2026

Protective effects of selenium against paclitaxel-induced testicular damage via modulation of oxidative stress, inflammation, and apoptotic pathways.

Protective effects of selenium against paclitaxel-induced testicular damage via modulation of oxidative stress, inflammation, and apoptotic pathways.

Journal: Reproductive toxicology (Elmsford, N.Y.)

Published: February 15, 2026

UROXIL attenuates cyclophosphamide-induced nephrotoxicity in rats by suppressing oxidative stress, endoplasmic reticulum stress, inflammation, and mitochondrial apoptosis.

UROXIL attenuates cyclophosphamide-induced nephrotoxicity in rats by suppressing oxidative stress, endoplasmic reticulum stress, inflammation, and mitochondrial apoptosis.

Journal: Tissue & cell

Published: February 05, 2026

Eugenol Mitigates Mercuric Chloride-Induced Renal Injury by Attenuating Oxidative Stress, Ferroptosis, ER Stress, Apoptosis, and Autophagy.

Eugenol Mitigates Mercuric Chloride-Induced Renal Injury by Attenuating Oxidative Stress, Ferroptosis, ER Stress, Apoptosis, and Autophagy.

Journal: Biological trace element research

Published: January 21, 2026

The neuroprotective role of eugenol against glyphosate-induced toxicity in rats: Modulation of oxidative stress, inflammation, ER stress and apoptotic signaling pathways.

The neuroprotective role of eugenol against glyphosate-induced toxicity in rats: Modulation of oxidative stress, inflammation, ER stress and apoptotic signaling pathways.

Journal: Tissue & cell

Published: December 19, 2025

Protective effects of p-coumaric acid against paclitaxel-induced testicular damage: Role of oxidative stress, inflammation, apoptosis, and autophagy.

Protective effects of p-coumaric acid against paclitaxel-induced testicular damage: Role of oxidative stress, inflammation, apoptosis, and autophagy.

Journal: Reproductive toxicology (Elmsford, N.Y.)

Published: December 06, 2025

Investigation of cellular pathophysiological differences in ER stress and apoptosis formation between classical and atypical forms of ovine pulmonary adenocarcinomas.

Investigation of cellular pathophysiological differences in ER stress and apoptosis formation between classical and atypical forms of ovine pulmonary adenocarcinomas.

Journal: Tissue & cell

Published: December 05, 2025

Hesperidin Attenuates Mercury Chloride-Induced Neurotoxicity in Rats by Modulating Oxidative Stress, Neuroinflammation, Apoptosis, Autophagy and ER Stress.

Hesperidin Attenuates Mercury Chloride-Induced Neurotoxicity in Rats by Modulating Oxidative Stress, Neuroinflammation, Apoptosis, Autophagy and ER Stress.

Journal: Biological trace element research

Published: November 28, 2025

Maedi infection modulates oncogenic and inflammatory signaling in ovine pulmonary adenomatosis through TLR4/NF-κB and JAK2/STAT3 pathways: A Trojan horse mechanism.

Maedi infection modulates oncogenic and inflammatory signaling in ovine pulmonary adenomatosis through TLR4/NF-κB and JAK2/STAT3 pathways: A Trojan horse mechanism.

Journal: Veterinary microbiology

Published: November 19, 2025

Showing 1-12 of 28

Last Updated: 09/26/2026