Michael Plotz

Berlin, BE, Germany

Last Updated: 09/26/2026

Save publications for later

Sign Up

Not sure about your diagnosis?

Check Your Symptoms

Found 12 publications

PI3K/AKT Signaling Pathway Is Essential for Survival of Induced Pluripotent Stem Cells.

PI3K/AKT Signaling Pathway Is Essential for Survival of Induced Pluripotent Stem Cells.

Journal: PloS one

Published: April 03, 2015

Sensitization of Melanoma Cells for Death Ligand TRAIL Is Based on Cell Cycle Arrest, ROS Production, and Activation of Proapoptotic Bcl-2 Proteins.

Sensitization of Melanoma Cells for Death Ligand TRAIL Is Based on Cell Cycle Arrest, ROS Production, and Activation of Proapoptotic Bcl-2 Proteins.

Journal: The Journal of investigative dermatology

Published: February 19, 2015

BH3-only proteins: possible proapoptotic triggers for melanoma therapy.

BH3-only proteins: possible proapoptotic triggers for melanoma therapy.

Journal: Experimental dermatology

Published: March 26, 2014

Sensitization of melanoma cells for TRAIL-induced apoptosis by activation of mitochondrial pathways via Bax.

Sensitization of melanoma cells for TRAIL-induced apoptosis by activation of mitochondrial pathways via Bax.

Journal: European journal of cell biology

Published: October 08, 2013

Synthesis and antiproliferative activity of selenoindirubins and selenoindirubin-N-glycosides.

Synthesis and antiproliferative activity of selenoindirubins and selenoindirubin-N-glycosides.

Journal: Organic & biomolecular chemistry

Published: May 16, 2013

Sensitization of melanoma cells for TRAIL-induced apoptosis by BMS-345541 correlates with altered phosphorylation and activation of Bax.

Sensitization of melanoma cells for TRAIL-induced apoptosis by BMS-345541 correlates with altered phosphorylation and activation of Bax.

Journal: Cell death & disease

Published: January 26, 2013

RAF inhibition overcomes resistance to TRAIL-induced apoptosis in melanoma cells.

RAF inhibition overcomes resistance to TRAIL-induced apoptosis in melanoma cells.

Journal: The Journal of investigative dermatology

Published: October 17, 2012

The BH3-only protein Bim(L) overrides Bcl-2-mediated apoptosis resistance in melanoma cells.

The BH3-only protein Bim(L) overrides Bcl-2-mediated apoptosis resistance in melanoma cells.

Journal: Cancer letters

Published: September 26, 2012

Disruption of the VDAC2-Bak interaction by Bcl-x(S) mediates efficient induction of apoptosis in melanoma cells.

Disruption of the VDAC2-Bak interaction by Bcl-x(S) mediates efficient induction of apoptosis in melanoma cells.

Journal: Cell death and differentiation

Published: June 19, 2012

Apoptosis induction by SAHA in cutaneous T-cell lymphoma cells is related to downregulation of c-FLIP and enhanced TRAIL signaling.

Apoptosis induction by SAHA in cutaneous T-cell lymphoma cells is related to downregulation of c-FLIP and enhanced TRAIL signaling.

Journal: The Journal of investigative dermatology

Published: May 04, 2012

Mutual regulation of Bcl-2 proteins independent of the BH3 domain as shown by the BH3-lacking protein Bcl-x(AK).

Mutual regulation of Bcl-2 proteins independent of the BH3 domain as shown by the BH3-lacking protein Bcl-x(AK).

Journal: PloS one

Published: November 03, 2011

Nonsteroidal anti-inflammatory drugs induce apoptosis in cutaneous T-cell lymphoma cells and enhance their sensitivity for TNF-related apoptosis-inducing ligand.

Nonsteroidal anti-inflammatory drugs induce apoptosis in cutaneous T-cell lymphoma cells and enhance their sensitivity for TNF-related apoptosis-inducing ligand.

Journal: The Journal of investigative dermatology

Published: October 21, 2011

Showing 1-12 of 12

Last Updated: 09/26/2026