Mickael Decressac
Lund, M 22184, Sweden
Last Updated: 09/26/2026
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Found 15 publications
Aged and Diseased Neurons Get Lost in Transport.
Journal: Trends in neurosciences
Published: February 08, 2016
Targeting α-synuclein: Therapeutic options.
Journal: Movement disorders : official journal of the Movement Disorder Society
Published: November 30, 2015
Nurr1 and Retinoid X Receptor Ligands Stimulate Ret Signaling in Dopamine Neurons and Can Alleviate α-Synuclein Disrupted Gene Expression.
Journal: The Journal of neuroscience : the official journal of the Society for Neuroscience
Published: October 23, 2015
Cyclosporin promotes neurorestoration and cell replacement therapy in pre-clinical models of Parkinson's disease.
Journal: Acta neuropathologica communications
Published: October 14, 2015
Alpha-Synuclein Produces Early Behavioral Alterations via Striatal Cholinergic Synaptic Dysfunction by Interacting With GluN2D N-Methyl-D-Aspartate Receptor Subunit.
Journal: Biological psychiatry
Published: May 05, 2015
NURR1 in Parkinson disease--from pathogenesis to therapeutic potential.
Journal: Nature reviews. Neurology
Published: October 16, 2013
mTOR inhibition alleviates L-DOPA-induced dyskinesia in parkinsonian rats.
Journal: Journal of Parkinson's disease
Published: August 14, 2013
TFEB-mediated autophagy rescues midbrain dopamine neurons from α-synuclein toxicity.
Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: April 24, 2013
Transcription factor Nurr1 maintains fiber integrity and nuclear-encoded mitochondrial gene expression in dopamine neurons.
Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: January 24, 2013
α-Synuclein-induced down-regulation of Nurr1 disrupts GDNF signaling in nigral dopamine neurons.
Journal: Science translational medicine
Published: December 11, 2012
Neuropeptide Y and its role in CNS disease and repair.
Journal: Experimental neurology
Published: July 06, 2012
Impaired neurotransmission caused by overexpression of α-synuclein in nigral dopamine neurons.
Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: February 09, 2012
Last Updated: 09/26/2026