Milena Paw

Krakow, MA 30387, Poland

Last Updated: 09/26/2026

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Found 22 publications

Fibroblasts: A Molecular and Pathophysiological Perspective.

Fibroblasts: A Molecular and Pathophysiological Perspective.

Journal: Biomedicines
Published: October 30, 2025

Editorial: Acta Biochimica Polonica reviews | 2023-2024.

Editorial: Acta Biochimica Polonica reviews | 2023-2024.

Journal: Acta biochimica Polonica
Published: October 16, 2025

Transcriptional orchestration of EMT: Unraveling novel molecular targets in pulmonary fibrosis.

Transcriptional orchestration of EMT: Unraveling novel molecular targets in pulmonary fibrosis.

Journal: Molecular therapy : the journal of the American Society of Gene Therapy
Published: October 07, 2024

Hypoxic extracellular vesicles from hiPSCs protect cardiomyocytes from oxidative damage by transferring antioxidant proteins and enhancing Akt/Erk/NRF2 signaling.

Hypoxic extracellular vesicles from hiPSCs protect cardiomyocytes from oxidative damage by transferring antioxidant proteins and enhancing Akt/Erk/NRF2 signaling.

Journal: Cell communication and signaling : CCS
Published: February 25, 2024

Bronchial Fibroblasts from Asthmatic Patients Display Impaired Responsiveness to Direct Current Electric Fields (dcEFs).

Bronchial Fibroblasts from Asthmatic Patients Display Impaired Responsiveness to Direct Current Electric Fields (dcEFs).

Journal: Biomedicines
Published: July 04, 2023

Hypoxia enhances anti-fibrotic properties of extracellular vesicles derived from hiPSCs via the miR302b-3p/TGFβ/SMAD2 axis.

Hypoxia enhances anti-fibrotic properties of extracellular vesicles derived from hiPSCs via the miR302b-3p/TGFβ/SMAD2 axis.

Journal: BMC medicine
Published: June 20, 2023

PPARδ Agonist GW501516 Suppresses the TGF-β-Induced Profibrotic Response of Human Bronchial Fibroblasts from Asthmatic Patients.

PPARδ Agonist GW501516 Suppresses the TGF-β-Induced Profibrotic Response of Human Bronchial Fibroblasts from Asthmatic Patients.

Journal: International journal of molecular sciences
Published: February 27, 2023

When healing turns into killing - the pathophysiology of pancreatic and hepatic fibrosis.

When healing turns into killing - the pathophysiology of pancreatic and hepatic fibrosis.

Journal: The Journal of physiology
Published: November 08, 2021

SB203580-A Potent p38 MAPK Inhibitor Reduces the Profibrotic Bronchial Fibroblasts Transition Associated with Asthma.

SB203580-A Potent p38 MAPK Inhibitor Reduces the Profibrotic Bronchial Fibroblasts Transition Associated with Asthma.

Journal: International journal of molecular sciences
Published: October 31, 2021

Time-extended exposure of gastric epithelial cells to secretome of Helicobacter pylori-activated fibroblasts induces reprogramming of gastric epithelium towards pre-cancerogenic and pro-invasive phenotype.

Time-extended exposure of gastric epithelial cells to secretome of Helicobacter pylori-activated fibroblasts induces reprogramming of gastric epithelium towards pre-cancerogenic and pro-invasive phenotype.

Journal: American journal of cancer research
Published: March 23, 2021

Asthma-derived fibroblast to myofibroblast transition is enhanced in comparison to fibroblasts derived from non-asthmatic patients in 3D in vitro culture due to Smad2/3 signalling.

Asthma-derived fibroblast to myofibroblast transition is enhanced in comparison to fibroblasts derived from non-asthmatic patients in 3D in vitro culture due to Smad2/3 signalling.

Journal: Acta biochimica Polonica
Published: July 01, 2020

Responsiveness of human bronchial fibroblasts and epithelial cells from asthmatic and non-asthmatic donors to the transforming growth factor-β1 in epithelial-mesenchymal trophic unit model.

Responsiveness of human bronchial fibroblasts and epithelial cells from asthmatic and non-asthmatic donors to the transforming growth factor-β1 in epithelial-mesenchymal trophic unit model.

Journal: BMC molecular and cell biology
Published: May 07, 2020
Showing 1-12 of 22

Last Updated: 09/26/2026