Mina Noura

Nagoya, Japan

Last Updated: 09/26/2026

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Found 20 publications

Statins induce monocytic differentiation in acute myeloid leukemia cells through the KLF4/DPYSL2A axis.

Statins induce monocytic differentiation in acute myeloid leukemia cells through the KLF4/DPYSL2A axis.

Journal: FEBS open bio
Published: May 09, 2025

Two-way inhibition of PAX5 transcriptional activity by PAX5::CBFA2T3.

Two-way inhibition of PAX5 transcriptional activity by PAX5::CBFA2T3.

Journal: FEBS open bio
Published: January 24, 2025

NUP98-BPTF promotes oncogenic transformation through PIM1 upregulation.

NUP98-BPTF promotes oncogenic transformation through PIM1 upregulation.

Journal: Cancer medicine
Published: January 19, 2024

RGS1 and CREB5 are direct and common transcriptional targets of ZNF384-fusion proteins.

RGS1 and CREB5 are direct and common transcriptional targets of ZNF384-fusion proteins.

Journal: Cancer medicine
Published: January 18, 2024

Suppression of super-enhancer-driven TAL1 expression by KLF4 in T-cell acute lymphoblastic leukemia.

Suppression of super-enhancer-driven TAL1 expression by KLF4 in T-cell acute lymphoblastic leukemia.

Journal: Oncogene
Published: June 15, 2023

Parbendazole as a promising drug for inducing differentiation of acute myeloid leukemia cells with various subtypes.

Parbendazole as a promising drug for inducing differentiation of acute myeloid leukemia cells with various subtypes.

Journal: Communications biology
Published: March 27, 2023

EBF1-JAK2 inhibits the PAX5 function through physical interaction with PAX5 and kinase activity.

EBF1-JAK2 inhibits the PAX5 function through physical interaction with PAX5 and kinase activity.

Journal: International journal of hematology
Published: December 25, 2022

Functional inhibition of MEF2 by C/EBP is a possible mechanism of leukemia development by CEBP-IGH fusion gene.

Functional inhibition of MEF2 by C/EBP is a possible mechanism of leukemia development by CEBP-IGH fusion gene.

Journal: Cancer science
Published: June 20, 2022

RUNX1-Survivin Axis Is a Novel Therapeutic Target for Malignant Rhabdoid Tumors.

RUNX1-Survivin Axis Is a Novel Therapeutic Target for Malignant Rhabdoid Tumors.

Journal: Molecules and cells
Published: November 23, 2021

Efficacy of a combination therapy targeting CDK4/6 and autophagy in a mouse xenograft model of t(8;21) acute myeloid leukemia.

Efficacy of a combination therapy targeting CDK4/6 and autophagy in a mouse xenograft model of t(8;21) acute myeloid leukemia.

Journal: Biochemistry and biophysics reports
Published: June 21, 2021

RUNX inhibitor suppresses graft-versus-host disease through targeting RUNX-NFATC2 axis.

RUNX inhibitor suppresses graft-versus-host disease through targeting RUNX-NFATC2 axis.

Journal: EJHaem
Published: April 28, 2021

Albendazole induces the terminal differentiation of acute myeloid leukaemia cells to monocytes by stimulating the Krüppel-like factor 4-dihydropyrimidinase-like 2A (KLF4-DPYSL2A) axis.

Albendazole induces the terminal differentiation of acute myeloid leukaemia cells to monocytes by stimulating the Krüppel-like factor 4-dihydropyrimidinase-like 2A (KLF4-DPYSL2A) axis.

Journal: British journal of haematology
Published: February 08, 2021
Showing 1-12 of 20

Last Updated: 09/26/2026