Mingchuan Liu
No. 127 West Changle Road
Xi'an, 710032, China
Xi'an, 710032, China
Last Updated: 09/26/2026
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Found 16 publications
LncRNA Airn preserves cardiomyocyte fatty acid oxidation to ameliorate diabetic cardiomyopathy by modulating the QKI-PPARα axis.
Journal: Redox biology
Published: July 20, 2026
Supramolecular antioxidant conductive injectable hydrogel encapsulating L-arginine for synergistic modulation of myocardial infarction.
Journal: Journal of controlled release : official journal of the Controlled Release Society
Published: August 07, 2025
Heart Regeneration and Repair: Molecular Mechanism and Therapeutic Targets.
Journal: MedComm
Published: September 05, 2024
Macrophage-derived extracellular vesicles represent a promising endogenous iron-chelating therapy for iron overload and cardiac injury in myocardial infarction.
Journal: Journal of nanobiotechnology
Published: March 19, 2024
Moderate-intensity interval exercise exacerbates cardiac lipotoxicity in high-fat, high-calories diet-fed mice.
Journal: Nature communications
Published: December 19, 2023
Elevated SLC40A1 impairs cardiac function and exacerbates mitochondrial dysfunction, oxidative stress, and apoptosis in ischemic myocardia.
Journal: International journal of biological sciences
Published: August 22, 2023
Reticulon 3 deficiency ameliorates post-myocardial infarction heart failure by alleviating mitochondrial dysfunction and inflammation.
Journal: MedComm
Published: August 22, 2023
Lipid overload-induced RTN3 activation leads to cardiac dysfunction by promoting lipid droplet biogenesis.
Journal: Cell death and differentiation
Published: March 07, 2023
Gut-microbiome-based predictive model for ST-elevation myocardial infarction in young male patients.
Gut-microbiome-based predictive model for ST-elevation myocardial infarction in young male patients.
Journal: Frontiers in microbiology
Published: August 30, 2022
N-glycosylation-mediated CD147 accumulation induces cardiac fibrosis in the diabetic heart through ALK5 activation.
Journal: International journal of biological sciences
Published: July 26, 2022
Cardiac-specific overexpression of Ndufs1 ameliorates cardiac dysfunction after myocardial infarction by alleviating mitochondrial dysfunction and apoptosis.
Journal: Experimental & molecular medicine
Published: November 20, 2021
Last Updated: 09/26/2026