Mingge Ding

15 Changlexi Road
Xi'an, 710032, China

Last Updated: 09/26/2026

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Found 28 publications

USP10 protects against pressure overload-induced mitochondrial morphofunctional defects and pathological cardiac hypertrophy through stabilizing cytoplasmic Mfn2.

USP10 protects against pressure overload-induced mitochondrial morphofunctional defects and pathological cardiac hypertrophy through stabilizing cytoplasmic Mfn2.

Journal: Redox biology

Published: May 18, 2025

O-GlcNAcylation-mediated endothelial metabolic memory contributes to cardiac damage via small extracellular vesicles.

O-GlcNAcylation-mediated endothelial metabolic memory contributes to cardiac damage via small extracellular vesicles.

Journal: Cell metabolism

Published: September 02, 2024

Association of Naples Prognostic Score with cardiovascular disease risk and its longitudinal prognostic impact on mortality in cardiovascular disease patients: Evidence from NHANES.

Association of Naples Prognostic Score with cardiovascular disease risk and its longitudinal prognostic impact on mortality in cardiovascular disease patients: Evidence from NHANES.

Journal: Nutrition, metabolism, and cardiovascular diseases : NMCD

Published: September 01, 2024

Alpha-ketoglutarate is required for chronic hypoxia-induced cardiac remodeling.

Alpha-ketoglutarate is required for chronic hypoxia-induced cardiac remodeling.

Journal: American journal of physiology. Cell physiology

Published: July 29, 2024

Letter by Chen et al Regarding Article, "Reduced Mitochondrial Protein Translation Promotes Cardiomyocyte Proliferation and Heart Regeneration".

Letter by Chen et al Regarding Article, "Reduced Mitochondrial Protein Translation Promotes Cardiomyocyte Proliferation and Heart Regeneration".

Journal: Circulation

Published: May 20, 2024

Letter by Li et al Regarding Article, "Jmjd4 Facilitates Pkm2 Degradation in Cardiomyocytes and Is Protective Against Dilated Cardiomyopathy".

Letter by Li et al Regarding Article, "Jmjd4 Facilitates Pkm2 Degradation in Cardiomyocytes and Is Protective Against Dilated Cardiomyopathy".

Journal: Circulation

Published: December 11, 2023

IP3R1-mediated MAMs formation contributes to mechanical trauma-induced hepatic injury and the protective effect of melatonin.

IP3R1-mediated MAMs formation contributes to mechanical trauma-induced hepatic injury and the protective effect of melatonin.

Journal: Cellular & molecular biology letters

Published: July 05, 2023

Ischemia/reperfusion-induced MiD51 upregulation recruits Drp1 to mitochondria and contributes to myocardial injury.

Ischemia/reperfusion-induced MiD51 upregulation recruits Drp1 to mitochondria and contributes to myocardial injury.

Journal: Biochemical and biophysical research communications

Published: April 10, 2023

Letter by Shi et al Regarding Article, "Proteasomal Degradation of TRAF2 Mediates Mitochondrial Dysfunction in Doxorubicin-Cardiomyopathy".

Letter by Shi et al Regarding Article, "Proteasomal Degradation of TRAF2 Mediates Mitochondrial Dysfunction in Doxorubicin-Cardiomyopathy".

Journal: Circulation

Published: March 27, 2023

Letter by De et al Regarding Article, "MicroRNA-210 Controls Mitochondrial Metabolism and Protects Heart Function in Myocardial Infarction".

Letter by De et al Regarding Article, "MicroRNA-210 Controls Mitochondrial Metabolism and Protects Heart Function in Myocardial Infarction".

Journal: Circulation

Published: September 19, 2022

Paeonol protects against doxorubicin-induced cardiotoxicity by promoting Mfn2-mediated mitochondrial fusion through activating the PKCε-Stat3 pathway.

Paeonol protects against doxorubicin-induced cardiotoxicity by promoting Mfn2-mediated mitochondrial fusion through activating the PKCε-Stat3 pathway.

Journal: Journal of advanced research

Published: April 22, 2022

Corrigendum to "Mfn2-mediated mitochondrial fusion alleviates doxorubicin-induced cardiotoxicity with enhancing its anticancer activity through metabolic switch" [Redox Biol. 52 (2022) 1-16/102311].

Corrigendum to "Mfn2-mediated mitochondrial fusion alleviates doxorubicin-induced cardiotoxicity with enhancing its anticancer activity through metabolic switch" [Redox Biol. 52 (2022) 1-16/102311].

Journal: Redox biology

Published: April 18, 2022

Showing 1-12 of 28

Last Updated: 09/26/2026