Minghao Qu

Shenzhen Branch
Shenzhen, 518120, China

Last Updated: 09/26/2026

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Found 13 publications

Sustained Hypoxia-Inducible Factor 1-Alpha Accumulation Disrupts the Articular Niche to Promote Osteoarthritis Pathogenesis.

Sustained Hypoxia-Inducible Factor 1-Alpha Accumulation Disrupts the Articular Niche to Promote Osteoarthritis Pathogenesis.

Journal: Aging cell
Published: April 03, 2026

Global prevalence, incidence, and years lived with disability (YLDs) of osteoarthritis: trends from 1990 to 2021 and projections to 2050.

Global prevalence, incidence, and years lived with disability (YLDs) of osteoarthritis: trends from 1990 to 2021 and projections to 2050.

Journal: Journal of orthopaedic translation
Published: June 26, 2025

Targeting Piezo1 channel to alleviate intervertebral disc degeneration.

Targeting Piezo1 channel to alleviate intervertebral disc degeneration.

Journal: Journal of orthopaedic translation
Published: July 30, 2024

Pip5k1γ promotes anabolism of nucleus pulposus cells and intervertebral disc homeostasis by activating CaMKII-Ampk pathway in aged mice.

Pip5k1γ promotes anabolism of nucleus pulposus cells and intervertebral disc homeostasis by activating CaMKII-Ampk pathway in aged mice.

Journal: Aging cell
Published: December 22, 2023

Comparison of Kindlin-2 deficiency-stimulated osteoarthritis-like lesions induced by Prg4CreERT2 versus AggrecanCreERT2 transgene in mice.

Comparison of Kindlin-2 deficiency-stimulated osteoarthritis-like lesions induced by Prg4CreERT2 versus AggrecanCreERT2 transgene in mice.

Journal: Journal of orthopaedic translation
Published: December 29, 2022

Loss of Pinch Proteins Causes Severe Degenerative Disc Disease-Like Lesions in Mice.

Loss of Pinch Proteins Causes Severe Degenerative Disc Disease-Like Lesions in Mice.

Journal: Aging and disease
Published: November 27, 2022

Osteoarthritis: pathogenic signaling pathways and therapeutic targets.

Osteoarthritis: pathogenic signaling pathways and therapeutic targets.

Journal: Signal transduction and targeted therapy
Published: September 15, 2022

Pip5k1c Loss in Chondrocytes Causes Spontaneous Osteoarthritic Lesions in Aged Mice.

Pip5k1c Loss in Chondrocytes Causes Spontaneous Osteoarthritic Lesions in Aged Mice.

Journal: Aging and disease
Published: June 01, 2022

Kindlin-2 loss in condylar chondrocytes causes spontaneous osteoarthritic lesions in the temporomandibular joint in mice.

Kindlin-2 loss in condylar chondrocytes causes spontaneous osteoarthritic lesions in the temporomandibular joint in mice.

Journal: International journal of oral science
Published: January 10, 2022

Kindlin-2 deletion in osteoprogenitors causes severe chondrodysplasia and low-turnover osteopenia in mice.

Kindlin-2 deletion in osteoprogenitors causes severe chondrodysplasia and low-turnover osteopenia in mice.

Journal: Journal of orthopaedic translation
Published: June 28, 2021

A Strigolactone Biosynthesis Gene Contributed to the Green Revolution in Rice.

A Strigolactone Biosynthesis Gene Contributed to the Green Revolution in Rice.

Journal: Molecular plant
Published: February 24, 2020

Karrikin Signaling Acts Parallel to and Additively with Strigolactone Signaling to Regulate Rice Mesocotyl Elongation in Darkness.

Karrikin Signaling Acts Parallel to and Additively with Strigolactone Signaling to Regulate Rice Mesocotyl Elongation in Darkness.

Journal: The Plant cell
Published: February 24, 2020
Showing 1-12 of 13

Last Updated: 09/26/2026