Mirco Muller

Carl Neuberg Str. 1
Hannover, NI 30625, Germany

Last Updated: 09/26/2026

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Found 8 publications

Serelaxin treatment promotes adaptive hypertrophy but does not prevent heart failure in experimental peripartum cardiomyopathy.

Serelaxin treatment promotes adaptive hypertrophy but does not prevent heart failure in experimental peripartum cardiomyopathy.

Journal: Cardiovascular research

Published: March 21, 2016

Low STAT3 expression sensitizes to toxic effects of β-adrenergic receptor stimulation in peripartum cardiomyopathy.

Low STAT3 expression sensitizes to toxic effects of β-adrenergic receptor stimulation in peripartum cardiomyopathy.

Journal: European heart journal

Published: July 02, 2015

Structure of the F-actin-tropomyosin complex.

Structure of the F-actin-tropomyosin complex.

Journal: Nature

Published: August 25, 2014

Molecular mechanisms of disease-related human β-actin mutations p.R183W and p.E364K.

Molecular mechanisms of disease-related human β-actin mutations p.R183W and p.E364K.

Journal: The FEBS journal

Published: May 13, 2014

The combined repetitive oligopeptides of clostridium difficile toxin A counteract premature cleavage of the glucosyl-transferase domain by stabilizing protein conformation.

The combined repetitive oligopeptides of clostridium difficile toxin A counteract premature cleavage of the glucosyl-transferase domain by stabilizing protein conformation.

Journal: Toxins

Published: April 22, 2014

Distinct functional interactions between actin isoforms and nonsarcomeric myosins.

Distinct functional interactions between actin isoforms and nonsarcomeric myosins.

Journal: PloS one

Published: May 13, 2013

Structure of the rigor actin-tropomyosin-myosin complex.

Structure of the rigor actin-tropomyosin-myosin complex.

Journal: Cell

Published: January 20, 2012

Functional characterization of the human α-cardiac actin mutations Y166C and M305L involved in hypertrophic cardiomyopathy.

Functional characterization of the human α-cardiac actin mutations Y166C and M305L involved in hypertrophic cardiomyopathy.

Journal: Cellular and molecular life sciences : CMLS

Published: December 22, 2011

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Last Updated: 09/26/2026