Mireille Daigle
451 Smyth Road
Ottawa, ON, Canada
Ottawa, ON, Canada
Last Updated: 09/26/2026
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Found 14 publications
Rapid reorganization of serotonin projections and antidepressant response to 5-HT1A-biased agonist NLX-101 in fluoxetine-resistant cF1ko mice.
Journal: Neuropharmacology
Published: August 02, 2024
CaMKIV-Mediated Phosphorylation Inactivates Freud-1/CC2D1A Repression for Calcium-Dependent 5-HT1A Receptor Gene Induction.
Journal: International journal of molecular sciences
Published: April 26, 2024
Chronic Desipramine Reverses Deficits in Cell Activity, Norepinephrine Innervation, and Anxiety-Depression Phenotypes in Fluoxetine-Resistant cF1ko Mice.
Journal: The Journal of neuroscience : the official journal of the Society for Neuroscience
Published: June 21, 2023
Fluoxetine-induced recovery of serotonin and norepinephrine projections in a mouse model of post-stroke depression.
Journal: Translational psychiatry
Published: January 13, 2020
Loss of Adult 5-HT1A Autoreceptors Results in a Paradoxical Anxiogenic Response to Antidepressant Treatment.
Journal: The Journal of neuroscience : the official journal of the Society for Neuroscience
Published: February 06, 2018
Loss of MeCP2 in adult 5-HT neurons induces 5-HT1A autoreceptors, with opposite sex-dependent anxiety and depression phenotypes.
Journal: Scientific reports
Published: December 20, 2017
Abrogated Freud-1/Cc2d1a Repression of 5-HT1A Autoreceptors Induces Fluoxetine-Resistant Anxiety/Depression-Like Behavior.
Journal: The Journal of neuroscience : the official journal of the Society for Neuroscience
Published: June 15, 2017
Concentration-Dependent Dual Mode of Zn Action at Serotonin 5-HT1A Receptors: In Vitro and In Vivo Studies.
Journal: Molecular neurobiology
Published: August 20, 2015
COMT polymorphism modulates the resting-state EEG alpha oscillatory response to acute nicotine in male non-smokers.
Journal: Genes, brain, and behavior
Published: February 19, 2015
Chronic mild stress and antidepressant treatment alter 5-HT1A receptor expression by modifying DNA methylation of a conserved Sp4 site.
Journal: Neurobiology of disease
Published: February 17, 2015
Last Updated: 09/26/2026