Misun Kwak

The mutual interaction of TRPC5 channel with polycystin proteins.

Journal: The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology

Published: August 06, 2024

Pore residues of transient receptor potential channels canonical 1 and 4 heteromer determine channel properties.

Journal: American journal of physiology. Cell physiology

Published: May 22, 2023

Comparison of characteristics of kick- and electric-scooter-related dental and maxillofacial injuries: A retrospective study.

Journal: Dental traumatology : official publication of International Association for Dental Traumatology

Published: April 26, 2023

Molecular architecture of the Gαi-bound TRPC5 ion channel.

Journal: Nature communications

Published: March 07, 2023

Negative self-regulation of transient receptor potential canonical 4 by the specific interaction with phospholipase C-δ1.

Journal: The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology

Published: November 26, 2022

TRPC5 channel instability induced by depalmitoylation protects striatal neurons against oxidative stress in Huntington's disease.

Journal: Biochimica et biophysica acta. Molecular cell research

Published: August 22, 2019

Identification of phospholipase C β downstream effect on transient receptor potential canonical 1/4, transient receptor potential canonical 1/5 channels.

Journal: The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology

Published: June 17, 2019

Identification of clustered phosphorylation sites in PKD2L1: how PKD2L1 channel activation is regulated by cyclic adenosine monophosphate signaling pathway.

Journal: Pflugers Archiv : European journal of physiology

Published: September 08, 2017

The mutual interaction of TRPC5 channel with polycystin proteins.

The mutual interaction of TRPC5 channel with polycystin proteins.

Pore residues of transient receptor potential channels canonical 1 and 4 heteromer determine channel properties.

Pore residues of transient receptor potential channels canonical 1 and 4 heteromer determine channel properties.

Comparison of characteristics of kick- and electric-scooter-related dental and maxillofacial injuries: A retrospective study.

Comparison of characteristics of kick- and electric-scooter-related dental and maxillofacial injuries: A retrospective study.

Molecular architecture of the Gαi-bound TRPC5 ion channel.

Molecular architecture of the Gαi-bound TRPC5 ion channel.

Negative self-regulation of transient receptor potential canonical 4 by the specific interaction with phospholipase C-δ1.

Negative self-regulation of transient receptor potential canonical 4 by the specific interaction with phospholipase C-δ1.

TRPC5 channel instability induced by depalmitoylation protects striatal neurons against oxidative stress in Huntington's disease.

TRPC5 channel instability induced by depalmitoylation protects striatal neurons against oxidative stress in Huntington's disease.

Identification of phospholipase C β downstream effect on transient receptor potential canonical 1/4, transient receptor potential canonical 1/5 channels.

Identification of phospholipase C β downstream effect on transient receptor potential canonical 1/4, transient receptor potential canonical 1/5 channels.

TRPC1 as a negative regulator for TRPC4 and TRPC5 channels.

TRPC1 as a negative regulator for TRPC4 and TRPC5 channels.

The role of calmodulin in regulating calcium-permeable PKD2L1 channel activity.

The role of calmodulin in regulating calcium-permeable PKD2L1 channel activity.

Dual action of the Gαq-PLCβ-PI(4,5)P2 pathway on TRPC1/4 and TRPC1/5 heterotetramers.

Dual action of the Gαq-PLCβ-PI(4,5)P2 pathway on TRPC1/4 and TRPC1/5 heterotetramers.

Gαi-mediated TRPC4 activation by polycystin-1 contributes to endothelial function via STAT1 activation.

Gαi-mediated TRPC4 activation by polycystin-1 contributes to endothelial function via STAT1 activation.

Identification of clustered phosphorylation sites in PKD2L1: how PKD2L1 channel activation is regulated by cyclic adenosine monophosphate signaling pathway.

Identification of clustered phosphorylation sites in PKD2L1: how PKD2L1 channel activation is regulated by cyclic adenosine monophosphate signaling pathway.