Mobeen Raja

Linz, 4040, Austria

Last Updated: 09/26/2026

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Found 19 publications

Mechanistic Insights into Protein Stability and Self-aggregation in GLUT1 Genetic Variants Causing GLUT1-Deficiency Syndrome.

Mechanistic Insights into Protein Stability and Self-aggregation in GLUT1 Genetic Variants Causing GLUT1-Deficiency Syndrome.

Journal: The Journal of membrane biology
Published: October 19, 2019

Identification of phlorizin binding domains in sodium-glucose cotransporter family: SGLT1 as a unique model system.

Identification of phlorizin binding domains in sodium-glucose cotransporter family: SGLT1 as a unique model system.

Journal: Biochimie
Published: March 29, 2015

Pathogenic mutations causing glucose transport defects in GLUT1 transporter: The role of intermolecular forces in protein structure-function.

Pathogenic mutations causing glucose transport defects in GLUT1 transporter: The role of intermolecular forces in protein structure-function.

Journal: Biophysical chemistry
Published: February 16, 2015

Special interaction of anionic phosphatidic acid promotes high secondary structure in tetrameric potassium channel.

Special interaction of anionic phosphatidic acid promotes high secondary structure in tetrameric potassium channel.

Journal: The Journal of membrane biology
Published: April 16, 2014

SLC5 and SLC2 transporters in epithelia-cellular role and molecular mechanisms.

SLC5 and SLC2 transporters in epithelia-cellular role and molecular mechanisms.

Journal: Current topics in membranes
Published: November 27, 2012

Structural insights into genetic variants of Na(+)/glucose cotransporter SGLT1 causing glucose-galactose malabsorption: vSGLT as a model structure.

Structural insights into genetic variants of Na(+)/glucose cotransporter SGLT1 causing glucose-galactose malabsorption: vSGLT as a model structure.

Journal: Cell biochemistry and biophysics
Published: March 03, 2012

Transferring knowledge towards understanding the pore stabilizing variations in K(+) channels: pore stability in K(+) channels.

Transferring knowledge towards understanding the pore stabilizing variations in K(+) channels: pore stability in K(+) channels.

Journal: Journal of bioenergetics and biomembranes
Published: February 22, 2012

The ATP-sensitive K(+) channel ABCC8 S1369A type 2 diabetes risk variant increases MgATPase activity.

The ATP-sensitive K(+) channel ABCC8 S1369A type 2 diabetes risk variant increases MgATPase activity.

Journal: Diabetes
Published: December 22, 2011

Diverse gating in K+ channels: differential role of the pore-helix glutamate in stabilizing the channel pore.

Diverse gating in K+ channels: differential role of the pore-helix glutamate in stabilizing the channel pore.

Journal: Biochemical and biophysical research communications
Published: July 28, 2011

Do small headgroups of phosphatidylethanolamine and phosphatidic acid lead to a similar folding pattern of the K(+) channel?

Do small headgroups of phosphatidylethanolamine and phosphatidic acid lead to a similar folding pattern of the K(+) channel?

Journal: The Journal of membrane biology
Published: April 28, 2011

Improved technique for reconstituting incredibly high and soluble amounts of tetrameric K⁺ channel in natural membranes.

Improved technique for reconstituting incredibly high and soluble amounts of tetrameric K⁺ channel in natural membranes.

Journal: The Journal of membrane biology
Published: March 25, 2011

The potassium channel KcsA: a model protein in studying membrane protein oligomerization and stability of oligomeric assembly?

The potassium channel KcsA: a model protein in studying membrane protein oligomerization and stability of oligomeric assembly?

Journal: Archives of biochemistry and biophysics
Published: December 31, 2010
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Last Updated: 09/26/2026