Mohammad Y. Alwetaid

Riyadh, 11671, Saudi Arabia

Last Updated: 09/26/2026

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Found 12 publications

Temporal and Regional Trends of Foodborne Diseases in Saudi Arabia (2015-2023): A Nationwide Retrospective Analysis.

Temporal and Regional Trends of Foodborne Diseases in Saudi Arabia (2015-2023): A Nationwide Retrospective Analysis.

Journal: Journal of epidemiology and global health

Published: September 05, 2025

Analysis of Suspected Food Poisoning Incidents in Riyadh, Saudi Arabia: An Epidemiological Perspective.

Analysis of Suspected Food Poisoning Incidents in Riyadh, Saudi Arabia: An Epidemiological Perspective.

Journal: International journal of microbiology

Published: May 12, 2025

Aflatoxin B1 exposure exacerbates chemokine receptor expression in the BTBR T+ Itpr3tf/J Mouse Model, unveiling insights into autism spectrum disorder: A focus on brain and spleen.

Aflatoxin B1 exposure exacerbates chemokine receptor expression in the BTBR T+ Itpr3tf/J Mouse Model, unveiling insights into autism spectrum disorder: A focus on brain and spleen.

Journal: Reproductive toxicology (Elmsford, N.Y.)

Published: March 02, 2024

Aflatoxin B1 exposure deteriorates immune abnormalities in a BTBR T+ Itpr3tf/J mouse model of autism by increasing inflammatory mediators' production in CD19-expressing cells.

Aflatoxin B1 exposure deteriorates immune abnormalities in a BTBR T+ Itpr3tf/J mouse model of autism by increasing inflammatory mediators' production in CD19-expressing cells.

Journal: Journal of neuroimmunology

Published: February 29, 2024

Cadmium exposure exacerbates immunological abnormalities in a BTBR T+ Itpr3tf/J autistic mouse model by upregulating inflammatory mediators in CD45R-expressing cells.

Cadmium exposure exacerbates immunological abnormalities in a BTBR T+ Itpr3tf/J autistic mouse model by upregulating inflammatory mediators in CD45R-expressing cells.

Journal: Journal of neuroimmunology

Published: October 29, 2023

The Exposure to Lead (Pb) Exacerbates Immunological Abnormalities in BTBR T+ Itpr3tf/J Mice through the Regulation of Signaling Pathways Relevant to T Cells.

The Exposure to Lead (Pb) Exacerbates Immunological Abnormalities in BTBR T+ Itpr3tf/J Mice through the Regulation of Signaling Pathways Relevant to T Cells.

Journal: International journal of molecular sciences

Published: September 24, 2023

Histamine H4 Receptor Antagonist Ameliorates the Progression of Experimental Autoimmune Encephalomyelitis via Regulation of T-Cell Imbalance.

Histamine H4 Receptor Antagonist Ameliorates the Progression of Experimental Autoimmune Encephalomyelitis via Regulation of T-Cell Imbalance.

Journal: International journal of molecular sciences

Published: September 06, 2023

Aflatoxin B1 Exposure Aggravates Neurobehavioral Deficits and Immune Dysfunctions of Th1, Th9, Th17, Th22, and T Regulatory Cell-Related Transcription Factor Signaling in the BTBR T+Itpr3tf/J Mouse Model of Autism.

Aflatoxin B1 Exposure Aggravates Neurobehavioral Deficits and Immune Dysfunctions of Th1, Th9, Th17, Th22, and T Regulatory Cell-Related Transcription Factor Signaling in the BTBR T+Itpr3tf/J Mouse Model of Autism.

Journal: Brain sciences

Published: August 30, 2023

Aflatoxin B1 Exacerbates Genomic Instability and Apoptosis in the BTBR Autism Mouse Model via Dysregulating DNA Repair Pathway.

Aflatoxin B1 Exacerbates Genomic Instability and Apoptosis in the BTBR Autism Mouse Model via Dysregulating DNA Repair Pathway.

Journal: Toxics

Published: June 25, 2023

DAPTA, a C-C Chemokine Receptor 5 (CCR5), Leads to the Downregulation of Notch/NF-κB Signaling and Proinflammatory Mediators in CD40+ Cells in Experimental Autoimmune Encephalomyelitis Model in SJL/J Mice.

DAPTA, a C-C Chemokine Receptor 5 (CCR5), Leads to the Downregulation of Notch/NF-κB Signaling and Proinflammatory Mediators in CD40+ Cells in Experimental Autoimmune Encephalomyelitis Model in SJL/J Mice.

Journal: Biomedicines

Published: April 09, 2023

Upregulation of Inflammatory Mediators in Peripheral Blood CD40+ Cells in Children with Autism Spectrum Disorder.

Upregulation of Inflammatory Mediators in Peripheral Blood CD40+ Cells in Children with Autism Spectrum Disorder.

Journal: International journal of molecular sciences

Published: March 19, 2023

β-1,3-Glucan reverses aflatoxin B1-mediated suppression of immune responses in mice.

β-1,3-Glucan reverses aflatoxin B1-mediated suppression of immune responses in mice.

Journal: Life sciences

Published: November 14, 2015

Showing 1-12 of 12

Last Updated: 09/26/2026