Moon-yong H. Cha

LG Chem Life Science R&D Campus
Seoul, Republic of Korea

Last Updated: 09/26/2026

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Found 19 publications

Aβ-induced mitochondrial dysfunction in neural progenitors controls KDM5A to influence neuronal differentiation.

Aβ-induced mitochondrial dysfunction in neural progenitors controls KDM5A to influence neuronal differentiation.

Journal: Experimental & molecular medicine

Published: July 25, 2021

Deep proteome profiling of the hippocampus in the 5XFAD mouse model reveals biological process alterations and a novel biomarker of Alzheimer's disease.

Deep proteome profiling of the hippocampus in the 5XFAD mouse model reveals biological process alterations and a novel biomarker of Alzheimer's disease.

Journal: Experimental & molecular medicine

Published: January 02, 2019

Nurr1 (NR4A2) regulates Alzheimer's disease-related pathogenesis and cognitive function in the 5XFAD mouse model.

Nurr1 (NR4A2) regulates Alzheimer's disease-related pathogenesis and cognitive function in the 5XFAD mouse model.

Journal: Aging cell

Published: August 21, 2018

Vitamin D-binding protein-loaded PLGA nanoparticles suppress Alzheimer's disease-related pathology in 5XFAD mice.

Vitamin D-binding protein-loaded PLGA nanoparticles suppress Alzheimer's disease-related pathology in 5XFAD mice.

Journal: Nanomedicine : nanotechnology, biology, and medicine

Published: August 11, 2018

Amyloid β-induced elevation of O-GlcNAcylated c-Fos promotes neuronal cell death.

Amyloid β-induced elevation of O-GlcNAcylated c-Fos promotes neuronal cell death.

Journal: Aging cell

Published: April 10, 2018

Retraction of "β-Amyloid and α-Synuclein Cooperate To Block SNARE-Dependent Vesicle Fusion".

Retraction of "β-Amyloid and α-Synuclein Cooperate To Block SNARE-Dependent Vesicle Fusion".

Journal: Biochemistry

Published: February 11, 2017

Close Correlation of Monoamine Oxidase Activity with Progress of Alzheimer's Disease in Mice, Observed by in Vivo Two-Photon Imaging.

Close Correlation of Monoamine Oxidase Activity with Progress of Alzheimer's Disease in Mice, Observed by in Vivo Two-Photon Imaging.

Journal: ACS central science

Published: October 18, 2016

Elimination of paternal mitochondria in mouse embryos occurs through autophagic degradation dependent on PARKIN and MUL1.

Elimination of paternal mitochondria in mouse embryos occurs through autophagic degradation dependent on PARKIN and MUL1.

Journal: eLife

Published: May 17, 2016

Insulin-degrading enzyme secretion from astrocytes is mediated by an autophagy-based unconventional secretory pathway in Alzheimer disease.

Insulin-degrading enzyme secretion from astrocytes is mediated by an autophagy-based unconventional secretory pathway in Alzheimer disease.

Journal: Autophagy

Published: March 11, 2016

Protein-Induced Pluripotent Stem Cells Ameliorate Cognitive Dysfunction and Reduce Aβ Deposition in a Mouse Model of Alzheimer's Disease.

Protein-Induced Pluripotent Stem Cells Ameliorate Cognitive Dysfunction and Reduce Aβ Deposition in a Mouse Model of Alzheimer's Disease.

Journal: Stem cells translational medicine

Published: February 11, 2016

Mitochondria-Targeting Ceria Nanoparticles as Antioxidants for Alzheimer's Disease.

Mitochondria-Targeting Ceria Nanoparticles as Antioxidants for Alzheimer's Disease.

Journal: ACS nano

Published: February 05, 2016

Mitochondrial ATP synthase activity is impaired by suppressed O-GlcNAcylation in Alzheimer's disease.

Mitochondrial ATP synthase activity is impaired by suppressed O-GlcNAcylation in Alzheimer's disease.

Journal: Human molecular genetics

Published: June 02, 2015

Showing 1-12 of 19

Last Updated: 09/26/2026