Mylene Ferrand

Lyon, France

Last Updated: 09/26/2026

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Found 9 publications

The nuclear receptor RXRA controls cellular senescence by regulating calcium signaling.

The nuclear receptor RXRA controls cellular senescence by regulating calcium signaling.

Journal: Aging cell
Published: January 02, 2018

Screening of a kinase library reveals novel pro-senescence kinases and their common NF-κB-dependent transcriptional program.

Screening of a kinase library reveals novel pro-senescence kinases and their common NF-κB-dependent transcriptional program.

Journal: Aging
Published: November 20, 2015

The PLA2R1-JAK2 pathway upregulates ERRα and its mitochondrial program to exert tumor-suppressive action.

The PLA2R1-JAK2 pathway upregulates ERRα and its mitochondrial program to exert tumor-suppressive action.

Journal: Oncogene
Published: July 07, 2015

Multidrug resistance protein 3 loss promotes tumor formation by inducing senescence escape.

Multidrug resistance protein 3 loss promotes tumor formation by inducing senescence escape.

Journal: Oncogene
Published: November 19, 2014

Repression of PLA2R1 by c-MYC and HIF-2alpha promotes cancer growth.

Repression of PLA2R1 by c-MYC and HIF-2alpha promotes cancer growth.

Journal: Oncotarget
Published: March 25, 2014

Endoplasmic reticulum calcium release through ITPR2 channels leads to mitochondrial calcium accumulation and senescence.

Endoplasmic reticulum calcium release through ITPR2 channels leads to mitochondrial calcium accumulation and senescence.

Journal: Nature communications
Published: November 26, 2013

PLA2R1 mediates tumor suppression by activating JAK2.

PLA2R1 mediates tumor suppression by activating JAK2.

Journal: Cancer research
Published: September 07, 2013

PLA2R1 kills cancer cells by inducing mitochondrial stress.

PLA2R1 kills cancer cells by inducing mitochondrial stress.

Journal: Free radical biology & medicine
Published: May 16, 2013

MicroRNA miR-30 family regulates non-attachment growth of breast cancer cells.

MicroRNA miR-30 family regulates non-attachment growth of breast cancer cells.

Journal: BMC genomics
Published: September 08, 2012
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Last Updated: 09/26/2026