Nabil Kaci
Paris, 75015, France
Last Updated: 09/26/2026
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Found 17 publications
Dkk1 inhibition restores mandibular growth in an achondroplasia mouse model.
Journal: Biology open
Published: February 24, 2026
Low-dose infigratinib increases bone growth and corrects growth plate abnormalities in an achondroplasia mouse model.
Journal: Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research
Published: November 10, 2023
Hypochondroplasia gain-of-function mutation in FGFR3 causes defective bone mineralization in mice.
Journal: JCI insight
Published: January 11, 2023
Prevention of guanylyl cyclase-B dephosphorylation rescues achondroplastic dwarfism.
Journal: JCI insight
Published: January 19, 2021
FGFR3 overactivation in the brain is responsible for memory impairments in Crouzon syndrome mouse model.
Journal: The Journal of experimental medicine
Published: September 01, 2020
Phosphatase inhibition by LB-100 enhances BMN-111 stimulation of bone growth.
Journal: JCI insight
Published: June 18, 2020
Constitutively-active FGFR3 disrupts primary cilium length and IFT20 trafficking in various chondrocyte models of achondroplasia.
Journal: Human molecular genetics
Published: August 18, 2017
Tyrosine kinase inhibitor NVP-BGJ398 functionally improves FGFR3-related dwarfism in mouse model.
Journal: The Journal of clinical investigation
Published: July 24, 2015
Last Updated: 09/26/2026