Nanshan Song

Nanjing, 210023, China

Last Updated: 09/26/2026

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Found 17 publications

Fibroblast growth factor homologous factors in neural signaling: Molecular mechanisms and disease-relevant dysregulation.

Fibroblast growth factor homologous factors in neural signaling: Molecular mechanisms and disease-relevant dysregulation.

Journal: Cytokine & growth factor reviews
Published: April 22, 2026

Complement receptor 3 (CR3)-dependent microglial synapse elimination drives Parkinson's disease pathogenesis in systemic inflammation.

Complement receptor 3 (CR3)-dependent microglial synapse elimination drives Parkinson's disease pathogenesis in systemic inflammation.

Journal: Cell death & disease
Published: March 24, 2025

Neuronal FGF13 Inhibits Mitochondria-Derived Damage Signals to Prevent Neuroinflammation and Neurodegeneration in a Mouse Model of Parkinson's Disease.

Neuronal FGF13 Inhibits Mitochondria-Derived Damage Signals to Prevent Neuroinflammation and Neurodegeneration in a Mouse Model of Parkinson's Disease.

Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)
Published: February 25, 2025

Antagonism of β-arrestins in IL-4-driven microglia reactivity via the Samd4/mTOR/OXPHOS axis in Parkinson's disease.

Antagonism of β-arrestins in IL-4-driven microglia reactivity via the Samd4/mTOR/OXPHOS axis in Parkinson's disease.

Journal: Science advances
Published: August 21, 2024

Focusing on mitochondria in the brain: from biology to therapeutics.

Focusing on mitochondria in the brain: from biology to therapeutics.

Journal: Translational neurodegeneration
Published: December 10, 2023

CircEPS15, as a sponge of MIR24-3p ameliorates neuronal damage in Parkinson disease through boosting PINK1-PRKN-mediated mitophagy.

CircEPS15, as a sponge of MIR24-3p ameliorates neuronal damage in Parkinson disease through boosting PINK1-PRKN-mediated mitophagy.

Journal: Autophagy
Published: April 04, 2023

Corrigendum: Induced expression of kir6.2 in A1 astrocytes propagates inflammatory neurodegeneration via Drp1-dependent mitochondrial fission.

Corrigendum: Induced expression of kir6.2 in A1 astrocytes propagates inflammatory neurodegeneration via Drp1-dependent mitochondrial fission.

Journal: Frontiers in pharmacology
Published: December 06, 2022

An imbalance of netrin-1 and DCC during nigral degeneration in experimental models and patients with Parkinson's disease.

An imbalance of netrin-1 and DCC during nigral degeneration in experimental models and patients with Parkinson's disease.

Journal: CNS neuroscience & therapeutics
Published: September 20, 2022

Impeding the combination of astrocytic ASCT2 and NLRP3 by talniflumate alleviates neuroinflammation in experimental models of Parkinson's disease.

Impeding the combination of astrocytic ASCT2 and NLRP3 by talniflumate alleviates neuroinflammation in experimental models of Parkinson's disease.

Journal: Acta pharmaceutica Sinica. B
Published: May 10, 2022

β-Arrestin2-biased Drd2 agonist UNC9995 alleviates astrocyte inflammatory injury via interaction between β-arrestin2 and STAT3 in mouse model of depression.

β-Arrestin2-biased Drd2 agonist UNC9995 alleviates astrocyte inflammatory injury via interaction between β-arrestin2 and STAT3 in mouse model of depression.

Journal: Journal of neuroinflammation
Published: February 27, 2022

Kir6.2 is essential to maintain neurite features by modulating PM20D1-reduced mitochondrial ATP generation.

Kir6.2 is essential to maintain neurite features by modulating PM20D1-reduced mitochondrial ATP generation.

Journal: Redox biology
Published: September 05, 2021

NLRP3/caspase-1/GSDMD-mediated pyroptosis exerts a crucial role in astrocyte pathological injury in mouse model of depression.

NLRP3/caspase-1/GSDMD-mediated pyroptosis exerts a crucial role in astrocyte pathological injury in mouse model of depression.

Journal: JCI insight
Published: December 14, 2020
Showing 1-12 of 17

Last Updated: 09/26/2026