Naokazu Sasagasako

Omuta, Japan

Last Updated: 09/26/2026

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Found 26 publications

Differential pathology of P102L-associated Gerstmann-Stäussler-Scheinker disease: exclusive presence of 8-kDa protease-resistant prion protein vs. co-existence of 8-kDa and type-1 protease-resistant prion protein, with a focus on codon 129 polymorphism.

Differential pathology of P102L-associated Gerstmann-Stäussler-Scheinker disease: exclusive presence of 8-kDa protease-resistant prion protein vs. co-existence of 8-kDa and type-1 protease-resistant prion protein, with a focus on codon 129 polymorphism.

Journal: Prion

Published: September 17, 2025

Plexin D1 accumulation in the spinal motor neurons of patients with amyotrophic lateral sclerosis.

Plexin D1 accumulation in the spinal motor neurons of patients with amyotrophic lateral sclerosis.

Journal: Journal of the neurological sciences

Published: October 11, 2024

Altered expression of human myxovirus resistance protein A in amyotrophic lateral sclerosis.

Altered expression of human myxovirus resistance protein A in amyotrophic lateral sclerosis.

Journal: Journal of neuropathology and experimental neurology

Published: June 25, 2024

Dopaminergic neurodegeneration in Gerstmann-Sträussler-Scheinker (P102L) disease: insights from imaging and pathological examination.

Dopaminergic neurodegeneration in Gerstmann-Sträussler-Scheinker (P102L) disease: insights from imaging and pathological examination.

Journal: Frontiers in neurology

Published: June 21, 2024

Novel method for classification of prion diseases by detecting PrPres signal patterns from formalin-fixed paraffin-embedded samples.

Novel method for classification of prion diseases by detecting PrPres signal patterns from formalin-fixed paraffin-embedded samples.

Journal: Prion

Published: April 16, 2024

Ribosomal protein SA is a common component of neuronal intranuclear inclusions in polyglutamine diseases and Marinesco bodies.

Ribosomal protein SA is a common component of neuronal intranuclear inclusions in polyglutamine diseases and Marinesco bodies.

Journal: Neuropathology : official journal of the Japanese Society of Neuropathology

Published: February 27, 2023

Mutated FUS in familial amyotrophic lateral sclerosis involves multiple hnRNPs in the formation of neuronal cytoplasmic inclusions.

Mutated FUS in familial amyotrophic lateral sclerosis involves multiple hnRNPs in the formation of neuronal cytoplasmic inclusions.

Journal: Journal of neuropathology and experimental neurology

Published: January 02, 2023

Increased expression of human antiviral protein MxA in FUS proteinopathy in amyotrophic lateral sclerosis.

Increased expression of human antiviral protein MxA in FUS proteinopathy in amyotrophic lateral sclerosis.

Journal: Brain pathology (Zurich, Switzerland)

Published: November 18, 2022

Neuropathology of classic myotonic dystrophy type 1 is characterized by both early initiation of primary age-related tauopathy of the hippocampus and unique 3-repeat tauopathy of the brainstem.

Neuropathology of classic myotonic dystrophy type 1 is characterized by both early initiation of primary age-related tauopathy of the hippocampus and unique 3-repeat tauopathy of the brainstem.

Journal: Journal of neuropathology and experimental neurology

Published: November 04, 2022

Silence of resident microglia in GPI anchorless prion disease and activation of microglia in Gerstmann-Sträussler-Scheinker disease and sporadic Creutzfeldt-Jakob disease.

Silence of resident microglia in GPI anchorless prion disease and activation of microglia in Gerstmann-Sträussler-Scheinker disease and sporadic Creutzfeldt-Jakob disease.

Journal: Journal of neuropathology and experimental neurology

Published: November 04, 2022

Chronological Changes in the Expression Pattern of Hippocampal Prion Proteins During Disease Progression in Sporadic Creutzfeldt-Jakob Disease MM1 Subtype.

Chronological Changes in the Expression Pattern of Hippocampal Prion Proteins During Disease Progression in Sporadic Creutzfeldt-Jakob Disease MM1 Subtype.

Journal: Journal of neuropathology and experimental neurology

Published: September 05, 2022

Early and extensive alterations of glial connexins, distal oligodendrogliopathy type demyelination, and nodal/paranodal pathology are characteristic of multiple system atrophy.

Early and extensive alterations of glial connexins, distal oligodendrogliopathy type demyelination, and nodal/paranodal pathology are characteristic of multiple system atrophy.

Journal: Brain pathology (Zurich, Switzerland)

Published: June 17, 2022

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Last Updated: 09/26/2026