Nathaniel Lal

Vancouver, BC, Canada

Last Updated: 09/26/2026

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Found 12 publications

Cardiac-specific VEGFB overexpression reduces lipoprotein lipase activity and improves insulin action in rat heart.

Cardiac-specific VEGFB overexpression reduces lipoprotein lipase activity and improves insulin action in rat heart.

Journal: American journal of physiology. Endocrinology and metabolism

Published: November 08, 2021

Involvement of Heparanase in Endothelial Cell-Cardiomyocyte Crosstalk.

Involvement of Heparanase in Endothelial Cell-Cardiomyocyte Crosstalk.

Journal: Advances in experimental medicine and biology

Published: April 11, 2020

Diabetes Mellitus Severity and a Switch From Using Lipoprotein Lipase to Adipose-Derived Fatty Acid Results in a Cardiac Metabolic Signature That Embraces Cell Death.

Diabetes Mellitus Severity and a Switch From Using Lipoprotein Lipase to Adipose-Derived Fatty Acid Results in a Cardiac Metabolic Signature That Embraces Cell Death.

Journal: Journal of the American Heart Association

Published: November 01, 2019

Vascular Endothelial Growth Factor B and Its Signaling.

Vascular Endothelial Growth Factor B and Its Signaling.

Journal: Frontiers in cardiovascular medicine

Published: January 29, 2018

Dual effects of hyperglycemia on endothelial cells and cardiomyocytes to enhance coronary LPL activity.

Dual effects of hyperglycemia on endothelial cells and cardiomyocytes to enhance coronary LPL activity.

Journal: American journal of physiology. Heart and circulatory physiology

Published: October 08, 2017

Loss of VEGFB and its signaling in the diabetic heart is associated with increased cell death signaling.

Loss of VEGFB and its signaling in the diabetic heart is associated with increased cell death signaling.

Journal: American journal of physiology. Heart and circulatory physiology

Published: September 29, 2016

Heparanase Overexpression Induces Glucagon Resistance and Protects Animals From Chemically Induced Diabetes.

Heparanase Overexpression Induces Glucagon Resistance and Protects Animals From Chemically Induced Diabetes.

Journal: Diabetes

Published: June 22, 2016

High glucose facilitated endothelial heparanase transfer to the cardiomyocyte modifies its cell death signature.

High glucose facilitated endothelial heparanase transfer to the cardiomyocyte modifies its cell death signature.

Journal: Cardiovascular research

Published: April 15, 2016

Cardiomyocyte VEGF Regulates Endothelial Cell GPIHBP1 to Relocate Lipoprotein Lipase to the Coronary Lumen During Diabetes Mellitus.

Cardiomyocyte VEGF Regulates Endothelial Cell GPIHBP1 to Relocate Lipoprotein Lipase to the Coronary Lumen During Diabetes Mellitus.

Journal: Arteriosclerosis, thrombosis, and vascular biology

Published: June 11, 2015

Endothelial cells respond to hyperglycemia by increasing the LPL transporter GPIHBP1.

Endothelial cells respond to hyperglycemia by increasing the LPL transporter GPIHBP1.

Journal: American journal of physiology. Endocrinology and metabolism

Published: April 17, 2014

Endothelial cell heparanase taken up by cardiomyocytes regulates lipoprotein lipase transfer to the coronary lumen after diabetes.

Endothelial cell heparanase taken up by cardiomyocytes regulates lipoprotein lipase transfer to the coronary lumen after diabetes.

Journal: Diabetes

Published: March 11, 2014

Hyperglycemia-induced secretion of endothelial heparanase stimulates a vascular endothelial growth factor autocrine network in cardiomyocytes that promotes recruitment of lipoprotein lipase.

Hyperglycemia-induced secretion of endothelial heparanase stimulates a vascular endothelial growth factor autocrine network in cardiomyocytes that promotes recruitment of lipoprotein lipase.

Journal: Arteriosclerosis, thrombosis, and vascular biology

Published: October 12, 2013

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Last Updated: 09/26/2026