Nicol Poncina

Padova, 35100, Italy

Last Updated: 09/26/2026

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Found 13 publications

p66Shc gene expression in peripheral blood mononuclear cells and progression of diabetic complications.

p66Shc gene expression in peripheral blood mononuclear cells and progression of diabetic complications.

Journal: Cardiovascular diabetology
Published: November 18, 2017

NETosis Delays Diabetic Wound Healing in Mice and Humans.

NETosis Delays Diabetic Wound Healing in Mice and Humans.

Journal: Diabetes
Published: June 24, 2015

Diabetes modifies the relationships among carotid plaque calcification, composition and inflammation.

Diabetes modifies the relationships among carotid plaque calcification, composition and inflammation.

Journal: Atherosclerosis
Published: April 13, 2015

p66Shc deletion or deficiency protects from obesity but not metabolic dysfunction in mice and humans.

p66Shc deletion or deficiency protects from obesity but not metabolic dysfunction in mice and humans.

Journal: Diabetologia
Published: February 19, 2015

Diabetes Limits Stem Cell Mobilization Following G-CSF but Not Plerixafor.

Diabetes Limits Stem Cell Mobilization Following G-CSF but Not Plerixafor.

Journal: Diabetes
Published: January 16, 2015

Bone Marrow Macrophages Contribute to Diabetic Stem Cell Mobilopathy by Producing Oncostatin M.

Bone Marrow Macrophages Contribute to Diabetic Stem Cell Mobilopathy by Producing Oncostatin M.

Journal: Diabetes
Published: September 27, 2014

NETosis is induced by high glucose and associated with type 2 diabetes.

NETosis is induced by high glucose and associated with type 2 diabetes.

Journal: Acta diabetologica
Published: September 10, 2014

The molecular signature of impaired diabetic wound healing identifies serpinB3 as a healing biomarker.

The molecular signature of impaired diabetic wound healing identifies serpinB3 as a healing biomarker.

Journal: Diabetologia
Published: April 22, 2014

The dipeptidyl peptidase-4 inhibitor saxagliptin improves function of circulating pro-angiogenic cells from type 2 diabetic patients.

The dipeptidyl peptidase-4 inhibitor saxagliptin improves function of circulating pro-angiogenic cells from type 2 diabetic patients.

Journal: Cardiovascular diabetology
Published: February 18, 2014

Diabetes causes bone marrow autonomic neuropathy and impairs stem cell mobilization via dysregulated p66Shc and Sirt1.

Diabetes causes bone marrow autonomic neuropathy and impairs stem cell mobilization via dysregulated p66Shc and Sirt1.

Journal: Diabetes
Published: November 26, 2013

Myeloid calcifying cells promote atherosclerotic calcification via paracrine activity and allograft inflammatory factor-1 overexpression.

Myeloid calcifying cells promote atherosclerotic calcification via paracrine activity and allograft inflammatory factor-1 overexpression.

Journal: Basic research in cardiology
Published: December 11, 2012

Diabetes impairs stem cell and proangiogenic cell mobilization in humans.

Diabetes impairs stem cell and proangiogenic cell mobilization in humans.

Journal: Diabetes care
Published: November 01, 2012
Showing 1-12 of 13

Last Updated: 09/26/2026