Nienke H. Van Teijlingen

Amsterdam, NH, Netherlands

Last Updated: 09/26/2026

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Found 15 publications

Prevotella timonensis Bacteria Associated With Vaginal Dysbiosis Enhance Human Immunodeficiency Virus Type 1 Susceptibility Of Vaginal CD4+ T Cells.

Prevotella timonensis Bacteria Associated With Vaginal Dysbiosis Enhance Human Immunodeficiency Virus Type 1 Susceptibility Of Vaginal CD4+ T Cells.

Journal: The Journal of infectious diseases
Published: October 26, 2023

Vaginal bacterium Prevotella timonensis turns protective Langerhans cells into HIV-1 reservoirs for virus dissemination.

Vaginal bacterium Prevotella timonensis turns protective Langerhans cells into HIV-1 reservoirs for virus dissemination.

Journal: The EMBO journal
Published: January 10, 2022

Autophagy-enhancing drugs limit mucosal HIV-1 acquisition and suppress viral replication ex vivo.

Autophagy-enhancing drugs limit mucosal HIV-1 acquisition and suppress viral replication ex vivo.

Journal: Scientific reports
Published: June 15, 2020

Sexually transmitted founder HIV-1 viruses are relatively resistant to Langerhans cell-mediated restriction.

Sexually transmitted founder HIV-1 viruses are relatively resistant to Langerhans cell-mediated restriction.

Journal: PloS one
Published: August 20, 2019

Vaginal dysbiosis associated-bacteria Megasphaera elsdenii and Prevotella timonensis induce immune activation via dendritic cells.

Vaginal dysbiosis associated-bacteria Megasphaera elsdenii and Prevotella timonensis induce immune activation via dendritic cells.

Journal: Journal of reproductive immunology
Published: August 15, 2019

Langerhans Cells Sense Staphylococcus aureus Wall Teichoic Acid through Langerin To Induce Inflammatory Responses.

Langerhans Cells Sense Staphylococcus aureus Wall Teichoic Acid through Langerin To Induce Inflammatory Responses.

Journal: mBio
Published: May 16, 2019

Immune activation of vaginal human Langerhans cells increases susceptibility to HIV-1 infection.

Immune activation of vaginal human Langerhans cells increases susceptibility to HIV-1 infection.

Journal: Scientific reports
Published: January 16, 2018

Erratum: HIV-1 blocks the signaling adaptor MAVS to evade antiviral host defense after sensing of abortive HIV-1 RNA by the host helicase DDX3.

Erratum: HIV-1 blocks the signaling adaptor MAVS to evade antiviral host defense after sensing of abortive HIV-1 RNA by the host helicase DDX3.

Journal: Nature immunology
Published: March 22, 2017

HIV-1 blocks the signaling adaptor MAVS to evade antiviral host defense after sensing of abortive HIV-1 RNA by the host helicase DDX3.

HIV-1 blocks the signaling adaptor MAVS to evade antiviral host defense after sensing of abortive HIV-1 RNA by the host helicase DDX3.

Journal: Nature immunology
Published: July 21, 2016

Probiotic Gut Microbiota Isolate Interacts with Dendritic Cells via Glycosylated Heterotrimeric Pili.

Probiotic Gut Microbiota Isolate Interacts with Dendritic Cells via Glycosylated Heterotrimeric Pili.

Journal: PloS one
Published: December 15, 2015

Cervicovaginal microbiome dysbiosis is associated with proteome changes related to alterations of the cervicovaginal mucosal barrier.

Cervicovaginal microbiome dysbiosis is associated with proteome changes related to alterations of the cervicovaginal mucosal barrier.

Journal: Mucosal immunology
Published: April 16, 2015

Selection of an HLA-C*03:04-Restricted HIV-1 p24 Gag Sequence Variant Is Associated with Viral Escape from KIR2DL3+ Natural Killer Cells: Data from an Observational Cohort in South Africa.

Selection of an HLA-C*03:04-Restricted HIV-1 p24 Gag Sequence Variant Is Associated with Viral Escape from KIR2DL3+ Natural Killer Cells: Data from an Observational Cohort in South Africa.

Journal: PLoS medicine
Published: August 12, 2014
Showing 1-12 of 15

Last Updated: 09/26/2026