Nils Welsh
Uppsala, C, Sweden
Last Updated: 09/26/2026
Save publications for later
Sign Up
Not sure about your diagnosis?
Check Your Symptoms
Found 38 publications
The Welander TIA1 mutation dedifferentiates insulin-producing cells: Reversal by a GLP-1 receptor agonist.
Journal: The Journal of biological chemistry
Published: December 09, 2025
AMP deaminase-2- and adenosine deaminase-mediated disposal of β-cell intracellular adenosine protects against multiple low-dose streptozotocin-induced type 1 diabetes.
Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie
Published: October 24, 2025
GDF15 Protects Insulin-Producing Beta Cells against Pro-Inflammatory Cytokines and Metabolic Stress via Increased Deamination of Intracellular Adenosine.
Journal: International journal of molecular sciences
Published: December 04, 2023
The selective NOX4 inhibitor GLX7013159 decreases blood glucose concentrations and human beta-cell apoptotic rates in diabetic NMRI nu/nu mice transplanted with human islets.
Journal: Free radical research
Published: November 16, 2023
CLEC11A improves insulin secretion and promotes cell proliferation in human beta-cells.
Journal: Journal of molecular endocrinology
Published: March 14, 2023
Metabolic stress-induced human beta-cell death is mediated by increased intracellular levels of adenosine.
Journal: Frontiers in endocrinology
Published: October 03, 2022
Are off-target effects of imatinib the key to improving beta-cell function in diabetes?
Journal: Upsala journal of medical sciences
Published: June 20, 2022
Pharmacological Inhibition of NOX4 Improves Mitochondrial Function and Survival in Human Beta-Cells.
Pharmacological Inhibition of NOX4 Improves Mitochondrial Function and Survival in Human Beta-Cells.
Journal: Biomedicines
Published: September 28, 2021
Imatinib protects against human beta-cell death via inhibition of mitochondrial respiration and activation of AMPK.
Journal: Clinical science (London, England : 1979)
Published: June 08, 2021
ZBED6 counteracts high-fat diet-induced glucose intolerance by maintaining beta cell area and reducing excess mitochondrial activation.
Journal: Diabetologia
Published: November 10, 2020
Last Updated: 09/26/2026