Niran Maharjan
Bern, BE, Switzerland
Last Updated: 09/26/2026
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Found 13 publications
Differential role of C-terminal truncations on alpha-synuclein pathology and Lewy body formation.
Journal: NPJ Parkinson's disease
Published: November 10, 2024
Sphingolipids and impaired hypoxic stress responses in Huntington disease.
Journal: Progress in lipid research
Published: November 21, 2022
Seeding the aggregation of TDP-43 requires post-fibrillization proteolytic cleavage.
Journal: Nature neuroscience
Published: May 24, 2022
PolyGA targets the ER stress-adaptive response by impairing GRP75 function at the MAM in C9ORF72-ALS/FTD.
Journal: Acta neuropathologica
Published: May 08, 2022
Emerging Perspectives on Dipeptide Repeat Proteins in C9ORF72 ALS/FTD.
Journal: Frontiers in cellular neuroscience
Published: December 03, 2020
Nuclear and cytoplasmic huntingtin inclusions exhibit distinct biochemical composition, interactome and ultrastructural properties.
Journal: Nature communications
Published: July 27, 2020
The process of Lewy body formation, rather than simply α-synuclein fibrillization, is one of the major drivers of neurodegeneration.
Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: February 21, 2020
ER strikes again: Proteostasis Dysfunction In ALS.
Journal: The EMBO journal
Published: March 13, 2016
C9ORF72 Regulates Stress Granule Formation and Its Deficiency Impairs Stress Granule Assembly, Hypersensitizing Cells to Stress.
Journal: Molecular neurobiology
Published: November 16, 2015
P2X1-regulated IL-22 secretion by innate lymphoid cells is required for efficient liver regeneration.
Journal: Hepatology (Baltimore, Md.)
Published: September 10, 2015
Aberrant association of misfolded SOD1 with Na(+)/K(+)ATPase-α3 impairs its activity and contributes to motor neuron vulnerability in ALS.
Journal: Acta neuropathologica
Published: August 23, 2015
Last Updated: 09/26/2026