Norio Uemura
Kumamoto, Japan
Last Updated: 09/26/2026
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Found 25 publications
Statins exert anti-growth effects by suppressing YAP/TAZ expressions via JNK signal activation and eliminate the immune suppression by downregulating PD-L1 expression in pancreatic cancer.
Journal: American journal of cancer research
Published: January 21, 2023
Biological and Clinical Impacts of Glucose Metabolism in Pancreatic Ductal Adenocarcinoma.
Journal: Cancers
Published: December 08, 2022
Hyperglycaemia induces metabolic reprogramming into a glycolytic phenotype and promotes epithelial-mesenchymal transitions via YAP/TAZ-Hedgehog signalling axis in pancreatic cancer.
Journal: British journal of cancer
Published: August 02, 2022
Cancer-associated fibroblasts reuse cancer-derived lactate to maintain a fibrotic and immunosuppressive microenvironment in pancreatic cancer.
Journal: JCI insight
Published: June 24, 2022
Thrombospondin-1 overexpression stimulates loss of Smad4 and accelerates malignant behavior via TGF-β signal activation in pancreatic ductal adenocarcinoma.
Journal: Translational oncology
Published: April 29, 2022
Frailty is associated with poor prognosis after resection for pancreatic cancer.
Journal: International journal of clinical oncology
Published: May 09, 2021
Statin as a therapeutic agent in gastroenterological cancer.
Journal: World journal of gastrointestinal oncology
Published: April 29, 2021
Biological Significance of YAP/TAZ in Pancreatic Ductal Adenocarcinoma.
Journal: Frontiers in oncology
Published: April 26, 2021
Proteomic Analysis of Malignant Ascites From Patients With Pancreatic Ductal Adenocarcinoma.
Journal: Anticancer research
Published: April 23, 2021
Risk factors for lymph node metastasis in patients with pancreatic neuroendocrine neoplasms.
Journal: World journal of clinical oncology
Published: April 10, 2021
Last Updated: 09/26/2026