Nuria L. Lorenzo
Santiago De Compostela, GA, Spain
Last Updated: 09/26/2026
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Found 10 publications
The L108I polymorphism in mouse prion protein drives spontaneous disease and enhances transmission of atypical and classical prion strains.
Journal: Brain pathology (Zurich, Switzerland)
Published: September 09, 2025
Spontaneous generation of diverse recombinant prion strains: sulfated glycan cofactors facilitate strain emergence but do not determine specific strain properties.
Journal: Acta neuropathologica communications
Published: June 12, 2025
Effect of primary structural variation on cervid prion protein in flexibility, stability, and spontaneous misfolding propensity.
Journal: Neurobiology of disease
Published: April 10, 2025
Conservation of strain properties of bank vole-adapted chronic wasting disease in the absence of glycosylation and membrane anchoring.
Journal: Neurobiology of disease
Published: January 29, 2025
Cofactors facilitate bona fide prion misfolding in vitro but are not necessary for the infectivity of recombinant murine prions.
Journal: PLoS pathogens
Published: October 04, 2024
Erratum: A tetracationic porphyrin with dual anti-prion activity.
Journal: iScience
Published: November 02, 2023
Understanding the key features of the spontaneous formation of bona fide prions through a novel methodology that enables their swift and consistent generation.
Journal: Acta neuropathologica communications
Published: June 29, 2023
Bona fide atypical scrapie faithfully reproduced for the first time in a rodent model.
Journal: Acta neuropathologica communications
Published: September 16, 2022
Pharmacological inactivation of the prion protein by targeting a folding intermediate.
Journal: Communications biology
Published: August 07, 2020
Showing 1-10 of 10
Last Updated: 09/26/2026