Olayemi J. Olajide
Ilorin, KW, Nigeria
Last Updated: 09/26/2026
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Found 28 publications
Perinatal walnut-enriched diet partially rescues mitochondrial dysfunction, neuroinflammation, and behavioral deficits induced by developmental manganese exposure.
Journal: The Journal of nutritional biochemistry
Published: September 16, 2025
Microglial senescence in neurodegeneration: Insights, implications, and therapeutic opportunities.
Journal: Neuroprotection (Chichester, England)
Published: October 04, 2024
Reduced 17β-estradiol following ovariectomy induces mitochondrial dysfunction and degradation of synaptic proteins in the entorhinal cortex.
Journal: Neuroscience
Published: July 19, 2024
Editorial: Neuroepigenetics and biological mechanisms of stress-induced socio-cognitive changes.
Journal: Frontiers in behavioral neuroscience
Published: February 07, 2024
Neuroinflammation and oxidative redox imbalance drive memory dysfunction in adolescent rats prenatally exposed to Datura Stramonium.
Journal: Neurotoxicology and teratology
Published: December 18, 2023
CRISPR-Cas9-mediated somatic correction of a one-base deletion in the Ugt1a gene ameliorates hyperbilirubinemia in Crigler-Najjar syndrome mice.
Journal: Molecular therapy. Methods & clinical development
Published: February 10, 2023
Activation of Alternative Bilirubin Clearance Pathways Partially Reduces Hyperbilirubinemia in a Mouse Model Lacking Functional Ugt1a1 Activity.
Journal: International journal of molecular sciences
Published: July 14, 2022
Inhibiting amyloid beta (1-42) peptide-induced mitochondrial dysfunction prevents the degradation of synaptic proteins in the entorhinal cortex.
Journal: Frontiers in aging neuroscience
Published: June 02, 2022
Suppression of Adenosine Deaminase and Xanthine Oxidase Activities by Mineralocorticoid and Glucocorticoid Receptor Blockades Restores Renal Antioxidative Barrier in Oral Contraceptive-Treated Dam.
Journal: Journal of the renin-angiotensin-aldosterone system : JRAAS
Published: March 11, 2021
Molecular mechanisms of neurodegeneration in the entorhinal cortex that underlie its selective vulnerability during the pathogenesis of Alzheimer's disease.
Journal: Biology open
Published: January 26, 2021
Amyloid-β (1-42) peptide induces rapid NMDA receptor-dependent alterations at glutamatergic synapses in the entorhinal cortex.
Journal: Neurobiology of aging
Published: January 14, 2021
Last Updated: 09/26/2026