Olena Shchyglo
Bochum, NW, Germany
Last Updated: 09/26/2026
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Found 9 publications
Intracerebral inoculation of healthy non-transgenic rats with a single aliquot of oligomeric amyloid-β (1-42) profoundly and progressively alters brain function throughout life.
Journal: Frontiers in aging neuroscience
Published: March 08, 2024
GluN2A or GluN2B subunits of the NMDA receptor contribute to changes in neuronal excitability and impairments in LTP in the hippocampus of aging mice but do not mediate detrimental effects of oligomeric Aβ (1-42).
Journal: Frontiers in aging neuroscience
Published: January 26, 2024
Changes in hippocampal volume, synaptic plasticity and amylin sensitivity in an animal model of type 2 diabetes are associated with increased vulnerability to amyloid-beta in advancing age.
Journal: Frontiers in aging neuroscience
Published: January 19, 2024
The Suprapyramidal and Infrapyramidal Blades of the Dentate Gyrus Exhibit Different GluN Subunit Content and Dissimilar Frequency-Dependent Synaptic Plasticity In Vivo.
Journal: Hippocampus
Published: January 05, 2024
Absence of Pannexin 1 Stabilizes Hippocampal Excitability After Intracerebral Treatment With Aβ (1-42) and Prevents LTP Deficits in Middle-Aged Mice.
Journal: Frontiers in aging neuroscience
Published: August 05, 2020
Hippocampal Synaptic Plasticity, Spatial Memory, and Neurotransmitter Receptor Expression Are Profoundly Altered by Gradual Loss of Hearing Ability.
Journal: Cerebral cortex (New York, N.Y. : 1991)
Published: October 28, 2019
Blocking VEGF by Bevacizumab Compromises Electrophysiological and Morphological Properties of Hippocampal Neurons.
Journal: Frontiers in cellular neuroscience
Published: November 21, 2018
Hippocampal function is compromised in an animal model of multiple sclerosis.
Journal: Neuroscience
Published: January 09, 2015
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Last Updated: 09/26/2026