Panagiotis Kavvadas

Paris, 75013, France

Last Updated: 09/26/2026

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Found 26 publications

Endothelial-specific deletion of connexin 43 improves renal function and structure after acute kidney injury.

Endothelial-specific deletion of connexin 43 improves renal function and structure after acute kidney injury.

Journal: Molecular medicine (Cambridge, Mass.)

Published: March 25, 2024

Signaling through cAMP-Epac1 induces metabolic reprogramming to protect podocytes in glomerulonephritis.

Signaling through cAMP-Epac1 induces metabolic reprogramming to protect podocytes in glomerulonephritis.

Journal: Kidney international

Published: September 22, 2023

Connexin-43 hemichannels orchestrate NOD-like receptor protein-3 (NLRP3) inflammasome activation and sterile inflammation in tubular injury.

Connexin-43 hemichannels orchestrate NOD-like receptor protein-3 (NLRP3) inflammasome activation and sterile inflammation in tubular injury.

Journal: Cell communication and signaling : CCS

Published: May 26, 2023

Deletion of Notch3 Impairs Contractility of Renal Resistance Vessels Due to Deficient Ca2+ Entry.

Deletion of Notch3 Impairs Contractility of Renal Resistance Vessels Due to Deficient Ca2+ Entry.

Journal: International journal of molecular sciences

Published: October 19, 2022

Activation of Notch3 in Renal Tubular Cells Leads to Progressive Cystic Kidney Disease.

Activation of Notch3 in Renal Tubular Cells Leads to Progressive Cystic Kidney Disease.

Journal: International journal of molecular sciences

Published: December 21, 2021

Notch3 expression in capillary pericytes predicts worse graft outcome in human renal grafts with antibody-mediated rejection.

Notch3 expression in capillary pericytes predicts worse graft outcome in human renal grafts with antibody-mediated rejection.

Journal: Journal of cellular and molecular medicine

Published: November 25, 2021

Down-regulation of human long non-coding RNA LINC01187 is associated with nephropathies.

Down-regulation of human long non-coding RNA LINC01187 is associated with nephropathies.

Journal: Journal of cellular and molecular medicine

Published: June 10, 2021

Endothelial-Specific Deletion of CD146 Protects Against Experimental Glomerulonephritis in Mice.

Endothelial-Specific Deletion of CD146 Protects Against Experimental Glomerulonephritis in Mice.

Journal: Hypertension (Dallas, Tex. : 1979)

Published: March 10, 2021

Super-resolved local recruitment of CLDN5 to filtration slits implicates a direct relationship with podocyte foot process effacement.

Super-resolved local recruitment of CLDN5 to filtration slits implicates a direct relationship with podocyte foot process effacement.

Journal: Journal of cellular and molecular medicine

Published: February 03, 2021

Blocking Connexin-43 mediated hemichannel activity protects against early tubular injury in experimental chronic kidney disease.

Blocking Connexin-43 mediated hemichannel activity protects against early tubular injury in experimental chronic kidney disease.

Journal: Cell communication and signaling : CCS

Published: September 12, 2019

Periostin Promotes Cell Proliferation and Macrophage Polarization to Drive Repair after AKI.

Periostin Promotes Cell Proliferation and Macrophage Polarization to Drive Repair after AKI.

Journal: Journal of the American Society of Nephrology : JASN

Published: February 05, 2019

MiR-21 is up-regulated in urinary exosomes of chronic kidney disease patients and after glomerular injury.

MiR-21 is up-regulated in urinary exosomes of chronic kidney disease patients and after glomerular injury.

Journal: Journal of cellular and molecular medicine

Published: November 05, 2018

Showing 1-12 of 26

Last Updated: 09/26/2026