Patricia Abbe

CNRS UMR7272, 28 Avenue Valrose
Nice, France

Last Updated: 09/26/2026

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Found 15 publications

Optimised arylbiamidine derivative as potent in vivo antimelanoma agent: Drug-to-target approach reveals nanomolar GSK3β inhibition.

Optimised arylbiamidine derivative as potent in vivo antimelanoma agent: Drug-to-target approach reveals nanomolar GSK3β inhibition.

Journal: Bioorganic chemistry

Published: January 10, 2025

Development and in vivo evaluation of fused benzazole analogs of anti-melanoma agent HA15.

Development and in vivo evaluation of fused benzazole analogs of anti-melanoma agent HA15.

Journal: Future medicinal chemistry

Published: June 07, 2021

Distinction between 2'- and 3'-Phosphate Isomers of a Fluorescent NADPH Analogue Led to Strong Inhibition of Cancer Cells Migration.

Distinction between 2'- and 3'-Phosphate Isomers of a Fluorescent NADPH Analogue Led to Strong Inhibition of Cancer Cells Migration.

Journal: Antioxidants (Basel, Switzerland)

Published: December 29, 2020

Dual Covalent Inhibition of PKM and IMPDH Targets Metabolism in Cutaneous Metastatic Melanoma.

Dual Covalent Inhibition of PKM and IMPDH Targets Metabolism in Cutaneous Metastatic Melanoma.

Journal: Cancer research

Published: June 22, 2020

Discovery of a new molecule inducing melanoma cell death: dual AMPK/MELK targeting for novel melanoma therapies.

Discovery of a new molecule inducing melanoma cell death: dual AMPK/MELK targeting for novel melanoma therapies.

Journal: Cell death & disease

Published: April 08, 2020

Innate lymphocyte-induced CXCR3B-mediated melanocyte apoptosis is a potential initiator of T-cell autoreactivity in vitiligo.

Innate lymphocyte-induced CXCR3B-mediated melanocyte apoptosis is a potential initiator of T-cell autoreactivity in vitiligo.

Journal: Nature communications

Published: September 27, 2018

E2F1 inhibition mediates cell death of metastatic melanoma.

E2F1 inhibition mediates cell death of metastatic melanoma.

Journal: Cell death & disease

Published: November 03, 2017

Structure activity relationship and optimization of N-(3-(2-aminothiazol-4-yl)aryl)benzenesulfonamides as anti-cancer compounds against sensitive and resistant cells.

Structure activity relationship and optimization of N-(3-(2-aminothiazol-4-yl)aryl)benzenesulfonamides as anti-cancer compounds against sensitive and resistant cells.

Journal: Bioorganic & medicinal chemistry letters

Published: January 20, 2017

Discovery and Optimization of N-(4-(3-Aminophenyl)thiazol-2-yl)acetamide as a Novel Scaffold Active against Sensitive and Resistant Cancer Cells.

Discovery and Optimization of N-(4-(3-Aminophenyl)thiazol-2-yl)acetamide as a Novel Scaffold Active against Sensitive and Resistant Cancer Cells.

Journal: Journal of medicinal chemistry

Published: August 31, 2016

Compounds Triggering ER Stress Exert Anti-Melanoma Effects and Overcome BRAF Inhibitor Resistance.

Compounds Triggering ER Stress Exert Anti-Melanoma Effects and Overcome BRAF Inhibitor Resistance.

Journal: Cancer cell

Published: August 02, 2016

Compounds Triggering ER Stress Exert Anti-Melanoma Effects and Overcome BRAF Inhibitor Resistance.

Compounds Triggering ER Stress Exert Anti-Melanoma Effects and Overcome BRAF Inhibitor Resistance.

Journal: Cancer cell

Published: July 23, 2015

Note: ultra-stable digitally controlled oven.

Note: ultra-stable digitally controlled oven.

Journal: The Review of scientific instruments

Published: March 02, 2015

Showing 1-12 of 15

Last Updated: 09/26/2026