Paula A. Deschamps
Hamilton, ON, Canada
Last Updated: 09/26/2026
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Found 9 publications
Deletion of transcription factor AP-2β from the developing murine trabecular meshwork region leads to progressive glaucomatous changes.
Journal: Journal of neuroscience research
Published: August 24, 2021
Conditional Deletion of AP-2β in the Periocular Mesenchyme of Mice Alters Corneal Epithelial Cell Fate and Stratification.
Journal: International journal of molecular sciences
Published: June 15, 2021
Progressive Loss of Retinal Ganglion Cells in Activating Protein-2β Neural Crest Cell Knockout Mice.
Progressive Loss of Retinal Ganglion Cells in Activating Protein-2β Neural Crest Cell Knockout Mice.
Journal: Current eye research
Published: March 10, 2021
AP-2β is required for formation of the murine trabecular meshwork and Schlemm's canal.
Journal: Experimental eye research
Published: February 06, 2020
Conditional Deletion of AP-2α and AP-2β in the Developing Murine Retina Leads to Altered Amacrine Cell Mosaics and Disrupted Visual Function.
Journal: Investigative ophthalmology & visual science
Published: May 02, 2018
β-Catenin/CBP-Dependent Signaling Regulates TGF-β-Induced Epithelial to Mesenchymal Transition of Lens Epithelial Cells.
Journal: Investigative ophthalmology & visual science
Published: October 28, 2016
Conditional deletion of AP-2β in mouse cranial neural crest results in anterior segment dysgenesis and early-onset glaucoma.
Journal: Disease models & mechanisms
Published: March 04, 2016
Matrix metalloproteinase-9-null mice are resistant to TGF-β-induced anterior subcapsular cataract formation.
Journal: The American journal of pathology
Published: June 21, 2013
Overlapping expression patterns and redundant roles for AP-2 transcription factors in the developing mammalian retina.
Journal: Developmental dynamics : an official publication of the American Association of Anatomists
Published: March 14, 2012
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Last Updated: 09/26/2026