Paula Marazuela

Barcelona, CT 08035, Spain

Last Updated: 09/26/2026

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Found 11 publications

The presence of circulating human apolipoprotein J reduces the occurrence of cerebral microbleeds in a transgenic mouse model with cerebral amyloid angiopathy.

The presence of circulating human apolipoprotein J reduces the occurrence of cerebral microbleeds in a transgenic mouse model with cerebral amyloid angiopathy.

Journal: Alzheimer's research & therapy

Published: April 29, 2024

Therapeutic effect of human ApoA-I-Milano variant in aged transgenic mouse model of Alzheimer's disease.

Therapeutic effect of human ApoA-I-Milano variant in aged transgenic mouse model of Alzheimer's disease.

Journal: British journal of pharmacology

Published: July 11, 2022

Impact of Cerebral Amyloid Angiopathy in Two Transgenic Mouse Models of Cerebral β-Amyloidosis: A Neuropathological Study.

Impact of Cerebral Amyloid Angiopathy in Two Transgenic Mouse Models of Cerebral β-Amyloidosis: A Neuropathological Study.

Journal: International journal of molecular sciences

Published: February 24, 2022

MFG-E8 (LACTADHERIN): a novel marker associated with cerebral amyloid angiopathy.

MFG-E8 (LACTADHERIN): a novel marker associated with cerebral amyloid angiopathy.

Journal: Acta neuropathologica communications

Published: September 02, 2021

Survival Bias and Crosstalk between Chronological and Behavioral Age: Age- and Genotype-Sensitivity Tests Define Behavioral Signatures in Middle-Aged, Old, and Long-Lived Mice with Normal and AD-Associated Aging.

Survival Bias and Crosstalk between Chronological and Behavioral Age: Age- and Genotype-Sensitivity Tests Define Behavioral Signatures in Middle-Aged, Old, and Long-Lived Mice with Normal and AD-Associated Aging.

Journal: Biomedicines

Published: May 05, 2021

Association of CD2AP neuronal deposits with Braak neurofibrillary stage in Alzheimer's disease.

Association of CD2AP neuronal deposits with Braak neurofibrillary stage in Alzheimer's disease.

Journal: Brain pathology (Zurich, Switzerland)

Published: March 08, 2021

Circulating AQP4 Levels in Patients with Cerebral Amyloid Angiopathy-Associated Intracerebral Hemorrhage.

Circulating AQP4 Levels in Patients with Cerebral Amyloid Angiopathy-Associated Intracerebral Hemorrhage.

Journal: Journal of clinical medicine

Published: January 07, 2021

Peripheral administration of human recombinant ApoJ/clusterin modulates brain beta-amyloid levels in APP23 mice.

Peripheral administration of human recombinant ApoJ/clusterin modulates brain beta-amyloid levels in APP23 mice.

Journal: Alzheimer's research & therapy

Published: December 19, 2018

CRISPR/Cas9-Correctable mutation-related molecular and physiological phenotypes in iPSC-derived Alzheimer's PSEN2 N141I neurons.

CRISPR/Cas9-Correctable mutation-related molecular and physiological phenotypes in iPSC-derived Alzheimer's PSEN2 N141I neurons.

Journal: Acta neuropathologica communications

Published: August 25, 2017

Characterization of ApoJ-reconstituted high-density lipoprotein (rHDL) nanodisc for the potential treatment of cerebral β-amyloidosis.

Characterization of ApoJ-reconstituted high-density lipoprotein (rHDL) nanodisc for the potential treatment of cerebral β-amyloidosis.

Journal: Scientific reports

Published: June 29, 2017

Intravenous treatment with human recombinant ApoA-I Milano reduces beta amyloid cerebral deposition in the APP23-transgenic mouse model of Alzheimer's disease.

Intravenous treatment with human recombinant ApoA-I Milano reduces beta amyloid cerebral deposition in the APP23-transgenic mouse model of Alzheimer's disease.

Journal: Neurobiology of aging

Published: February 27, 2017

Showing 1-11 of 11

Last Updated: 09/26/2026