Pierluigi Saba

Cagliari, Italy

Last Updated: 09/26/2026

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Found 12 publications

Combined α2- and D2-receptor blockade activates noradrenergic and dopaminergic neurons, but extracellular dopamine in the prefrontal cortex is determined by uptake and release from noradrenergic terminals.

Combined α2- and D2-receptor blockade activates noradrenergic and dopaminergic neurons, but extracellular dopamine in the prefrontal cortex is determined by uptake and release from noradrenergic terminals.

Journal: Frontiers in pharmacology

Published: June 10, 2023

The potent α2-adrenoceptor antagonist RS 79948 also inhibits dopamine D2 -receptors: Comparison with atipamezole and raclopride.

The potent α2-adrenoceptor antagonist RS 79948 also inhibits dopamine D2 -receptors: Comparison with atipamezole and raclopride.

Journal: Neuropharmacology

Published: November 03, 2021

Prenatal THC Does Not Affect Female Mesolimbic Dopaminergic System in Preadolescent Rats.

Prenatal THC Does Not Affect Female Mesolimbic Dopaminergic System in Preadolescent Rats.

Journal: International journal of molecular sciences

Published: January 07, 2021

Noradrenergic Source of Dopamine Assessed by Microdialysis in the Medial Prefrontal Cortex.

Noradrenergic Source of Dopamine Assessed by Microdialysis in the Medial Prefrontal Cortex.

Journal: Frontiers in pharmacology

Published: July 28, 2020

Prenatal THC exposure produces a hyperdopaminergic phenotype rescued by pregnenolone.

Prenatal THC exposure produces a hyperdopaminergic phenotype rescued by pregnenolone.

Journal: Nature neuroscience

Published: September 14, 2018

Noradrenergic terminals are the primary source of α2-adrenoceptor mediated dopamine release in the medial prefrontal cortex.

Noradrenergic terminals are the primary source of α2-adrenoceptor mediated dopamine release in the medial prefrontal cortex.

Journal: Progress in neuro-psychopharmacology & biological psychiatry

Published: July 23, 2018

Levodopa prevents the reinstatement of cocaine self-administration in rats via potentiation of dopamine release in the medial prefrontal cortex.

Levodopa prevents the reinstatement of cocaine self-administration in rats via potentiation of dopamine release in the medial prefrontal cortex.

Journal: Addiction biology

Published: August 05, 2016

The neurosteroidogenic enzyme 5α-reductase modulates the role of D1 dopamine receptors in rat sensorimotor gating.

The neurosteroidogenic enzyme 5α-reductase modulates the role of D1 dopamine receptors in rat sensorimotor gating.

Journal: Psychoneuroendocrinology

Published: June 23, 2015

Selective inhibition of dopamine-beta-hydroxylase enhances dopamine release from noradrenergic terminals in the medial prefrontal cortex.

Selective inhibition of dopamine-beta-hydroxylase enhances dopamine release from noradrenergic terminals in the medial prefrontal cortex.

Journal: Brain and behavior

Published: April 27, 2015

Elevated dopamine in the medial prefrontal cortex suppresses cocaine seeking via D1 receptor overstimulation.

Elevated dopamine in the medial prefrontal cortex suppresses cocaine seeking via D1 receptor overstimulation.

Journal: Addiction biology

Published: August 20, 2014

Inhibition of 17α-hydroxylase/C17,20 lyase reduces gating deficits consequent to dopaminergic activation.

Inhibition of 17α-hydroxylase/C17,20 lyase reduces gating deficits consequent to dopaminergic activation.

Journal: Psychoneuroendocrinology

Published: June 07, 2013

The dopamine beta-hydroxylase inhibitor nepicastat increases dopamine release and potentiates psychostimulant-induced dopamine release in the prefrontal cortex.

The dopamine beta-hydroxylase inhibitor nepicastat increases dopamine release and potentiates psychostimulant-induced dopamine release in the prefrontal cortex.

Journal: Addiction biology

Published: January 08, 2013

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Last Updated: 09/26/2026