Pinelopi P. Kapitsinou

Nephrology

675 N St Clair St Ste 18-250
Galter Pavilion
Chicago, IL 60611

Last Updated: 09/26/2026

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Found 20 publications

Enabling organ- and injury-specific nanocarrier targeting via surface-functionalized PEG-b-PPS micelles for acute kidney injury.

Enabling organ- and injury-specific nanocarrier targeting via surface-functionalized PEG-b-PPS micelles for acute kidney injury.

Journal: Nanoscale horizons

Published: October 06, 2025

Enabling Organ- and Injury-Specific Nanocarrier Targeting via Surface-Functionalized PEG-b-PPS Micelles for Acute Kidney Injury.

Enabling Organ- and Injury-Specific Nanocarrier Targeting via Surface-Functionalized PEG-b-PPS Micelles for Acute Kidney Injury.

Journal: bioRxiv : the preprint server for biology

Published: August 12, 2025

Endothelial prolyl hydroxylase 3 mitigates maladaptive inflammation to promote post-ischemic kidney repair.

Endothelial prolyl hydroxylase 3 mitigates maladaptive inflammation to promote post-ischemic kidney repair.

Journal: Kidney international

Published: May 19, 2025

Fueling kidney recovery: boosting BCAA metabolism to overcome nephrotoxic AKI.

Fueling kidney recovery: boosting BCAA metabolism to overcome nephrotoxic AKI.

Journal: American journal of physiology. Renal physiology

Published: March 06, 2025

Metabolic Breakdown: The Hidden Role of Endothelial Glycolysis in Kidney Fibrosis.

Metabolic Breakdown: The Hidden Role of Endothelial Glycolysis in Kidney Fibrosis.

Journal: Journal of the American Society of Nephrology : JASN

Published: November 27, 2024

SGLT2 inhibition protects kidney function by SAM-dependent epigenetic repression of inflammatory genes under metabolic stress.

SGLT2 inhibition protects kidney function by SAM-dependent epigenetic repression of inflammatory genes under metabolic stress.

Journal: The Journal of clinical investigation

Published: November 12, 2024

Post-ischemic inactivation of HIF prolyl hydroxylases in endothelium promotes maladaptive kidney repair by inducing glycolysis.

Post-ischemic inactivation of HIF prolyl hydroxylases in endothelium promotes maladaptive kidney repair by inducing glycolysis.

Journal: bioRxiv : the preprint server for biology

Published: October 24, 2023

Postischemic inactivation of HIF prolyl hydroxylases in endothelium promotes maladaptive kidney repair by inducing glycolysis.

Postischemic inactivation of HIF prolyl hydroxylases in endothelium promotes maladaptive kidney repair by inducing glycolysis.

Journal: The Journal of clinical investigation

Published: October 02, 2023

Chemical inhibition of oxygen-sensing prolyl hydroxylases impairs angiogenic competence of human vascular endothelium through metabolic reprogramming.

Chemical inhibition of oxygen-sensing prolyl hydroxylases impairs angiogenic competence of human vascular endothelium through metabolic reprogramming.

Journal: iScience

Published: March 25, 2022

Role of Endothelial Prolyl-4-Hydroxylase Domain Protein/Hypoxia-Inducible Factor Axis in Acute Kidney Injury.

Role of Endothelial Prolyl-4-Hydroxylase Domain Protein/Hypoxia-Inducible Factor Axis in Acute Kidney Injury.

Journal: Nephron

Published: April 09, 2021

Hypoxic preconditioning protects against ischemic kidney injury through the IDO1/kynurenine pathway.

Hypoxic preconditioning protects against ischemic kidney injury through the IDO1/kynurenine pathway.

Journal: Cell reports

Published: September 10, 2020

Iron Deficiency in Chronic Kidney Disease: Updates on Pathophysiology, Diagnosis, and Treatment.

Iron Deficiency in Chronic Kidney Disease: Updates on Pathophysiology, Diagnosis, and Treatment.

Journal: Journal of the American Society of Nephrology : JASN

Published: February 12, 2020

Showing 1-12 of 20

Last Updated: 09/26/2026