Prerana Sarangi

Balasore, OR 756003, India

Last Updated: 09/26/2026

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Found 10 publications

Activation of the MKK4/7-JNK-c-Jun axis mediates bisphenol F-induced neurodegeneration and behavioural alteration in adult zebrafish.

Activation of the MKK4/7-JNK-c-Jun axis mediates bisphenol F-induced neurodegeneration and behavioural alteration in adult zebrafish.

Journal: Environmental toxicology and pharmacology

Published: December 23, 2025

Bisphenol S-Induced Neurobehavioral Impairment Is Characterized by c-Jun Activation and Distinct Dysregulation of Nrf2 and BDNF in the Zebrafish Brain.

Bisphenol S-Induced Neurobehavioral Impairment Is Characterized by c-Jun Activation and Distinct Dysregulation of Nrf2 and BDNF in the Zebrafish Brain.

Journal: Neurotoxicity research

Published: December 15, 2025

Targeting the Microbiota-Gut-Brain Axis: Bacillus coagulans Protects Against Rotenone-Induced Parkinson's Disease in Rats.

Targeting the Microbiota-Gut-Brain Axis: Bacillus coagulans Protects Against Rotenone-Induced Parkinson's Disease in Rats.

Journal: Neurochemical research

Published: December 14, 2025

Attenuation of rotenone-induced neurodegeneration by Bacillus coagulans supplementation in zebrafish.

Attenuation of rotenone-induced neurodegeneration by Bacillus coagulans supplementation in zebrafish.

Journal: Comparative biochemistry and physiology. Toxicology & pharmacology : CBP

Published: August 03, 2025

Chronic exposure to bisphenol S is associated with antagonistic neurobehavioral transformation and cleaved caspase 3 induced neurodegeneration in zebrafish brain.

Chronic exposure to bisphenol S is associated with antagonistic neurobehavioral transformation and cleaved caspase 3 induced neurodegeneration in zebrafish brain.

Journal: Comparative biochemistry and physiology. Toxicology & pharmacology : CBP

Published: December 11, 2024

Bisphenol F-induced precocious genesis of aggressive neurobehavioral response is associated with heightened monoamine oxidase activity and neurodegeneration in zebrafish brain.

Bisphenol F-induced precocious genesis of aggressive neurobehavioral response is associated with heightened monoamine oxidase activity and neurodegeneration in zebrafish brain.

Journal: Neurotoxicology and teratology

Published: June 28, 2024

Concerted monoamine oxidase activity following exposure to di-2-ethylhexyl phthalate is associated with aggressive neurobehavioral response and neurodegeneration in zebrafish brain.

Concerted monoamine oxidase activity following exposure to di-2-ethylhexyl phthalate is associated with aggressive neurobehavioral response and neurodegeneration in zebrafish brain.

Journal: Comparative biochemistry and physiology. Toxicology & pharmacology : CBP

Published: April 18, 2024

Bisphenol A-induced neurobehavioral transformation is associated with augmented monoamine oxidase activity and neurodegeneration in zebrafish brain.

Bisphenol A-induced neurobehavioral transformation is associated with augmented monoamine oxidase activity and neurodegeneration in zebrafish brain.

Journal: Environmental toxicology and pharmacology

Published: September 28, 2022

Di-2-ethylhexyl phthalate-induced neurobehavioural transformation is associated with altered glutathione biosynthesis and neurodegeneration in zebrafish brain.

Di-2-ethylhexyl phthalate-induced neurobehavioural transformation is associated with altered glutathione biosynthesis and neurodegeneration in zebrafish brain.

Journal: Fish physiology and biochemistry

Published: September 12, 2022

Suppression of Chronic Unpredictable Stress-Persuaded Increased Monoamine Oxidase Activity by Taurine Promotes Significant Neuroprotection in Zebrafish Brain.

Suppression of Chronic Unpredictable Stress-Persuaded Increased Monoamine Oxidase Activity by Taurine Promotes Significant Neuroprotection in Zebrafish Brain.

Journal: Neurochemical research

Published: June 17, 2022

Showing 1-10 of 10

Last Updated: 09/26/2026