Priscila C. Campos
Belo Horizonte, MG, Brazil
Last Updated: 09/26/2026
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Found 18 publications
Galectin-3 regulates proinflammatory cytokine function and favours Brucella abortus chronic replication in macrophages and mice.
Journal: Cellular microbiology
Published: February 17, 2021
NLRP6-associated host microbiota composition impacts in the intestinal barrier to systemic dissemination of Brucella abortus.
Journal: PLoS neglected tropical diseases
Published: September 13, 2020
The Role of ST2 Receptor in the Regulation of Brucella abortus Oral Infection.
Journal: Pathogens (Basel, Switzerland)
Published: January 27, 2020
Guanylate-binding proteins at the crossroad of noncanonical inflammasome activation during bacterial infections.
Journal: Journal of leukocyte biology
Published: January 15, 2019
Brucella abortus nitric oxide metabolite regulates inflammasome activation and IL-1β secretion in murine macrophages.
Journal: European journal of immunology
Published: November 19, 2018
Brucella abortus Cyclic Dinucleotides Trigger STING-Dependent Unfolded Protein Response That Favors Bacterial Replication.
Journal: Journal of immunology (Baltimore, Md. : 1950)
Published: September 06, 2018
IL-1R and Inflammasomes Mediate Early Pulmonary Protective Mechanisms in Respiratory Brucella Abortus Infection.
Journal: Frontiers in cellular and infection microbiology
Published: August 01, 2018
The cGAS/STING Pathway Is Important for Dendritic Cell Activation but Is Not Essential to Induce Protective Immunity against Mycobacterium tuberculosis Infection.
Journal: Journal of innate immunity
Published: January 31, 2018
Immunoproteasome Subunits Are Required for CD8+ T Cell Function and Host Resistance to Brucella abortus Infection in Mice.
Journal: Infection and immunity
Published: August 27, 2017
TLR7 and TLR3 Sense Brucella abortus RNA to Induce Proinflammatory Cytokine Production but They Are Dispensable for Host Control of Infection.
Journal: Frontiers in immunology
Published: November 16, 2016
NLRP12 negatively regulates proinflammatory cytokine production and host defense against Brucella abortus.
Journal: European journal of immunology
Published: April 28, 2016
TLR9 is required for MAPK/NF-κB activation but does not cooperate with TLR2 or TLR6 to induce host resistance to Brucella abortus.
Journal: Journal of leukocyte biology
Published: August 06, 2015
Last Updated: 09/26/2026