Pu-hyeon Cha

Seoul, Republic of Korea

Last Updated: 09/26/2026

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Found 21 publications

Development of stromal corneal grafts using a novel decellularization method with sodium cocoyl glutamate on GGTA1/CMAH/β4GalNT2 knock-out porcine corneas.

Development of stromal corneal grafts using a novel decellularization method with sodium cocoyl glutamate on GGTA1/CMAH/β4GalNT2 knock-out porcine corneas.

Journal: Annals of translational medicine

Published: January 04, 2025

Destabilization of β-catenin and RAS by targeting the Wnt/β-catenin pathway as a potential treatment for triple-negative breast cancer.

Destabilization of β-catenin and RAS by targeting the Wnt/β-catenin pathway as a potential treatment for triple-negative breast cancer.

Journal: Experimental & molecular medicine

Published: March 03, 2020

APC loss induces Warburg effect via increased PKM2 transcription in colorectal cancer.

APC loss induces Warburg effect via increased PKM2 transcription in colorectal cancer.

Journal: British journal of cancer

Published: February 24, 2020

A mutant KRAS-induced factor REG4 promotes cancer stem cell properties via Wnt/β-catenin signaling.

A mutant KRAS-induced factor REG4 promotes cancer stem cell properties via Wnt/β-catenin signaling.

Journal: International journal of cancer

Published: June 17, 2019

Indirubin-3'-oxime stimulates chondrocyte maturation and longitudinal bone growth via activation of the Wnt/β-catenin pathway.

Indirubin-3'-oxime stimulates chondrocyte maturation and longitudinal bone growth via activation of the Wnt/β-catenin pathway.

Journal: Experimental & molecular medicine

Published: April 11, 2019

CXXC5 mediates growth plate senescence and is a target for enhancement of longitudinal bone growth.

CXXC5 mediates growth plate senescence and is a target for enhancement of longitudinal bone growth.

Journal: Life science alliance

Published: November 23, 2018

A small molecule approach to degrade RAS with EGFR repression is a potential therapy for KRAS mutation-driven colorectal cancer resistance to cetuximab.

A small molecule approach to degrade RAS with EGFR repression is a potential therapy for KRAS mutation-driven colorectal cancer resistance to cetuximab.

Journal: Experimental & molecular medicine

Published: June 22, 2018

A Ras destabilizer KYA1797K overcomes the resistance of EGFR tyrosine kinase inhibitor in KRAS-mutated non-small cell lung cancer.

A Ras destabilizer KYA1797K overcomes the resistance of EGFR tyrosine kinase inhibitor in KRAS-mutated non-small cell lung cancer.

Journal: Scientific reports

Published: June 01, 2018

β-Catenin-RAS interaction serves as a molecular switch for RAS degradation via GSK3β.

β-Catenin-RAS interaction serves as a molecular switch for RAS degradation via GSK3β.

Journal: EMBO reports

Published: March 05, 2018

Stabilization of Sur8 via PKCα/δ degradation promotes transformation and migration of colorectal cancer cells.

Stabilization of Sur8 via PKCα/δ degradation promotes transformation and migration of colorectal cancer cells.

Journal: Oncotarget

Published: July 15, 2017

KY1022, a small molecule destabilizing Ras via targeting the Wnt/β-catenin pathway, inhibits development of metastatic colorectal cancer.

KY1022, a small molecule destabilizing Ras via targeting the Wnt/β-catenin pathway, inhibits development of metastatic colorectal cancer.

Journal: Oncotarget

Published: August 20, 2016

Simultaneous destabilization of β-catenin and Ras via targeting of the axin-RGS domain as a potential therapeutic strategy for colorectal cancer.

Simultaneous destabilization of β-catenin and Ras via targeting of the axin-RGS domain as a potential therapeutic strategy for colorectal cancer.

Journal: BMB reports

Published: July 26, 2016

Showing 1-12 of 21

Last Updated: 09/26/2026